DOES THE MITOCHONDRIAL-DNA PLAY A ROLE IN THE PATHOGENESIS OF DIABETES

被引:72
作者
GERBITZ, KD
机构
[1] Institutes of Clinical Chemistry and Diabetes Research, Krankenhaus München-Schwabing, Munich
关键词
D O I
10.1007/BF00401375
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
[No abstract available]
引用
收藏
页码:1181 / 1186
页数:6
相关论文
共 60 条
  • [31] DETERMINANTS OF THE SELECTIVE TOXICITY OF ALLOXAN TO THE PANCREATIC B-CELL
    MALAISSE, WJ
    MALAISSELAGAE, F
    SENER, A
    PIPELEERS, DG
    [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1982, 79 (03): : 927 - 930
  • [32] RECOMBINATION VIA FLANKING DIRECT REPEATS IS A MAJOR CAUSE OF LARGE-SCALE DELETIONS OF HUMAN MITOCHONDRIAL-DNA
    MITA, S
    RIZZUTO, R
    MORAES, CT
    SHANSKE, S
    ARNAUDO, E
    FABRIZI, GM
    KOGA, Y
    DIMAURO, S
    SCHON, EA
    [J]. NUCLEIC ACIDS RESEARCH, 1990, 18 (03) : 561 - 567
  • [33] MONCADA S, 1991, PHARMACOL REV, V43, P109
  • [34] MURPHY AE, 1975, PRINCIPLES GENETIC C, P343
  • [35] DIFFERENT COPY NUMBERS OF APPARENTLY IDENTICALLY DELETED MITOCHONDRIAL-DNA IN TISSUES FROM A PATIENT WITH KEARNS-SAYRE SYNDROME DETECTED BY PCR
    OBERMAIERKUSSER, B
    MULLERHOCKER, J
    NELSON, I
    LESTIENNE, P
    ENTER, C
    RIEDELE, T
    GERBITZ, KD
    [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1990, 169 (03) : 1007 - 1015
  • [36] Okamoto H., 2008, MOL BIOL ISLETS LANG, P209
  • [37] PATIENTS WITH IDIOPATHIC CARDIOMYOPATHY BELONG TO THE SAME MITOCHONDRIAL-DNA GENE FAMILY OF PARKINSONS-DISEASE AND MITOCHONDRIAL ENCEPHALOMYOPATHY
    OZAWA, T
    TANAKA, M
    SUGIYAMA, S
    INO, H
    OHNO, K
    HATTORI, K
    OHBAYASHI, T
    ITO, T
    DEGUCHI, H
    KAWAMURA, K
    NAKANE, Y
    HASHIBA, K
    [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1991, 177 (01) : 518 - 525
  • [38] PETTEPHER CC, 1991, J BIOL CHEM, V266, P3113
  • [39] QUADE A, 1988, KLIN WOCHENSCHR, V66, P272
  • [40] DO MITOCHONDRIAL-DNA FRAGMENTS PROMOTE CANCER AND AGING
    RICHTER, C
    [J]. FEBS LETTERS, 1988, 241 (1-2) : 1 - 5