INVITRO ANALYSIS OF MODULATORS OF INTERCELLULAR COMMUNICATION - IMPLICATIONS FOR BIOLOGICALLY BASED RISK ASSESSMENT MODELS FOR CHEMICAL-EXPOSURE

被引：32

作者：

TROSKO, JE

CHANG, CC

MADHUKAR, BV

机构：

[1] Department of Pediatrics/Human Development, Center for Environmental Toxicology, Michigan State University, East Lansing

来源：

TOXICOLOGY IN VITRO | 1990年 / 4卷 / 4-5期

关键词：

D O I：

10.1016/0887-2333(90)90133-E

中图分类号：

R99 [毒物学（毒理学）];

学科分类号：

100405 ;

摘要：

[No abstract available]

引用

页码：635 / 643

页数：9

共 113 条

[41] ENHANCEMENT OF TRANSFORMATION AND CONTINUOUS INHIBITION OF INTERCELLULAR COMMUNICATION BY 1-OLEOYL-2-ACETYL GLYCEROL IN BALB/C 3T3-CELLS [J].

FRIXEN, U ;

YAMASAKI, H .

CARCINOGENESIS, 1987, 8 (08) :1101-1104

[42] DIACYLGLYCEROL INHIBITS GAP JUNCTIONAL COMMUNICATION IN CULTURED EPIDERMAL-CELLS - EVIDENCE FOR A ROLE OF PROTEIN KINASE-C [J].

GAINER, HS ;

MURRAY, AW .

BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1985, 126 (03) :1109-1113

[43] SERTOLI CELL OCCLUDING JUNCTIONS AND GAP JUNCTIONS IN MATURE AND DEVELOPING MAMMALIAN TESTIS [J].

GILULA, NB ;

FAWCETT, DW ;

AOKI, A .

DEVELOPMENTAL BIOLOGY, 1976, 50 (01) :142-168

[44] THE HYPOTHETICAL WAY OF PROGRESS [J].

HUSZAGH, VA ;

INFANTE, JP .

NATURE, 1989, 338 (6211) :109-109

[45] THE RELEVANCE OF GAP-JUNCTIONS TO STAGE I TUMOR PROMOTION IN MOUSE EPIDERMIS [J].

KALIMI, GH ;

SIRSAT, SM .

CARCINOGENESIS, 1984, 5 (12) :1671-1677

[46] MODULATION OF CELL COMMUNICATION AND CARCINOGENESIS [J].

KANNO, Y .

JAPANESE JOURNAL OF PHYSIOLOGY, 1985, 35 (05) :693-707

[47]

KLANN RC, 1989, CANCER RES, V49, P699

[48] THE APPROACHING ERA OF THE TUMOR SUPPRESSOR GENES [J].

KLEIN, G .

SCIENCE, 1987, 238 (4833) :1539-1545

[49]

KNUDSON AG, 1985, CANCER RES, V45, P1437

[50] PERFORMANCE OF SHORT-TERM TESTS FOR DETECTION OF HUMAN CARCINOGENS [J].

KUROKI, T ;

MATSUSHIMA, T .

MUTAGENESIS, 1987, 2 (01) :33-37

← 1 2 3 4 5 6 7 8 9 10 →