血糖波动与氧化应激

被引:13
作者
李红
李霖
机构
[1] 浙江大学医学院附属邵逸夫医院内分泌科
关键词
D O I
暂无
中图分类号
R686 [筋腱、韧带、滑囊疾病及损伤];
学科分类号
100220 [骨科学];
摘要
<正> 传统观点认为糖尿病并发症由晚期糖基化终产物(AGEs)、多元醇通路、蛋白激酶C(PKC)、己糖胺途径激活所致,而Brownlee证明线粒体活性氧簇(ROS)生成过多可能是糖尿病并发症发生的共同机制。良好的血糖控制是延缓或阻止糖尿病慢性并发
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