Akt与细胞线粒体凋亡途径

[1] 重组人红细胞生成素对癫痫持续状态大鼠海马p-Akt和caspase-9的影响及作用机制[J]. 王维平,史志勤,于江华,郭力,王乐,韩东亮,袁栋才,臧颖卓.南方医科大学学报. 2010(01)

[2] Bax BH3结构域的多肽链(H2-H3)通过抑制Bcl-2的成孔和中和Bcl-2的抗Bax作用而引起细胞凋亡(英文) [J]. 生物医学工程学杂志 , 2009, (04) : 829 - 835

[3] Evaluation of the Role of Hexokinase Type II in Cellular Proliferation and Apoptosis Using Human Hepatocellular Carcinoma Cell Lines [J]. JOURNAL OF NUCLEAR MEDICINE, 2009, 50 (09) : 1525 - 1532

[4] Inhibiting PI3K as a therapeutic strategy against cancer[J] . Luis Paz-Ares,Carmen Blanco-Aparicio,Rocío García-Carbonero,Amancio Carnero.Clinical and Translational Oncology . 2009 (9)

[5] The nuclear localization of 3′-phosphoinositide-dependent kinase-1 is dependent on its association with the protein tyrosine phosphatase SHP-1 [J]. CELLULAR SIGNALLING, 2009, 21 (11) : 1634 - 1644

[6] A critical role for AKT activation in protecting cells from ionizing radiation-induced apoptosis and the regulation of acinus gene expression [J]. EUROPEAN JOURNAL OF CELL BIOLOGY, 2009, 88 (10) : 563 - 575

[7] c-Jun N-Terminal Kinase Regulates the Interaction Between 14-3-3 and Bad in Ethanol-Induced Cell Death [J]. JOURNAL OF NEUROSCIENCE RESEARCH, 2008, 86 (14) : 3221 - 3229

[8] Overexpression of phospho-Akt correlates with phosphorylation of EGFreceptor, FKHR and BAD in nasopharyngeal carcinoma[J] . Wai Yip,Vincent Leong,Maizaton Abdullah,Suryati Yusoff,Heng Seow.Oncology Reports . 2008 (2)

[9] The major apoptotic endonuclease DFF40/CAD is a deoxyribose-specific and double-strand-specific enzyme [J]. APOPTOSIS, 2008, 13 (03) : 377 - 382

[10] BimL displacing Bcl-xL promotes bax translocation during TNFα-induced apoptosis [J]. APOPTOSIS, 2008, 13 (07) : 950 - 958

[1] 重组人红细胞生成素对癫痫持续状态大鼠海马p-Akt和caspase-9的影响及作用机制[J]. 王维平,史志勤,于江华,郭力,王乐,韩东亮,袁栋才,臧颖卓.南方医科大学学报. 2010(01)

[2] Bax BH3结构域的多肽链(H2-H3)通过抑制Bcl-2的成孔和中和Bcl-2的抗Bax作用而引起细胞凋亡(英文) [J]. 生物医学工程学杂志 , 2009, (04) : 829 - 835

[3] Evaluation of the Role of Hexokinase Type II in Cellular Proliferation and Apoptosis Using Human Hepatocellular Carcinoma Cell Lines [J]. JOURNAL OF NUCLEAR MEDICINE, 2009, 50 (09) : 1525 - 1532

[4] Inhibiting PI3K as a therapeutic strategy against cancer[J] . Luis Paz-Ares,Carmen Blanco-Aparicio,Rocío García-Carbonero,Amancio Carnero.Clinical and Translational Oncology . 2009 (9)

[5] The nuclear localization of 3′-phosphoinositide-dependent kinase-1 is dependent on its association with the protein tyrosine phosphatase SHP-1 [J]. CELLULAR SIGNALLING, 2009, 21 (11) : 1634 - 1644

[6] A critical role for AKT activation in protecting cells from ionizing radiation-induced apoptosis and the regulation of acinus gene expression [J]. EUROPEAN JOURNAL OF CELL BIOLOGY, 2009, 88 (10) : 563 - 575

[7] c-Jun N-Terminal Kinase Regulates the Interaction Between 14-3-3 and Bad in Ethanol-Induced Cell Death [J]. JOURNAL OF NEUROSCIENCE RESEARCH, 2008, 86 (14) : 3221 - 3229

[8] Overexpression of phospho-Akt correlates with phosphorylation of EGFreceptor, FKHR and BAD in nasopharyngeal carcinoma[J] . Wai Yip,Vincent Leong,Maizaton Abdullah,Suryati Yusoff,Heng Seow.Oncology Reports . 2008 (2)

[9] The major apoptotic endonuclease DFF40/CAD is a deoxyribose-specific and double-strand-specific enzyme [J]. APOPTOSIS, 2008, 13 (03) : 377 - 382

[10] BimL displacing Bcl-xL promotes bax translocation during TNFα-induced apoptosis [J]. APOPTOSIS, 2008, 13 (07) : 950 - 958