白癜风发病机制及治疗靶点的研究现状

被引:17
作者
许爱娥
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[1] 杭州市第三人民医院皮肤科
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R758.41 [];
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[1]
Transcriptome analysis reveals markers of aberrantly activated innate immunity in vitiligo lesional and non-lesional skin..[J].Richard Yu;Raewyn Broady;Yuanshen Huang;Yang Wang;Jie Yu;Min Gao;Megan Levings;Shencai Wei;Shengquan Zhang;Aie Xu;Mingwan Su;Jan Dutz;Xuejun Zhang;Youwen Zhou.PLoS ONE.2017, 12
[2]
Oxidative stress-induced overexpression of miR-25: the mechanism underlying the degeneration of melanocytes in vitiligo [J].
Shi, Q. ;
Zhang, W. ;
Guo, S. ;
Jian, Z. ;
Li, S. ;
Li, K. ;
Ge, R. ;
Dai, W. ;
Wang, G. ;
Gao, T. ;
Li, C. .
CELL DEATH AND DIFFERENTIATION, 2016, 23 (03) :496-508
[3]
Rapid skin repigmentation on oral ruxolitinib in a patient with coexistent vitiligo and alopecia areata (AA) [J].
Harris, John E. ;
Rashighi, Mehdi ;
Nhan Nguyen ;
Jabbari, Ali ;
Ulerio, Grace ;
Clynes, Raphael ;
Christiano, Angela M. ;
Mackay-Wiggan, Julian .
JOURNAL OF THE AMERICAN ACADEMY OF DERMATOLOGY, 2016, 74 (02) :370-371
[4]
The role of IL-17 in vitiligo: A review [J].
Singh, Rasnik K. ;
Lee, Kristina M. ;
Vujkovic-Cvijin, Ivan ;
Ucmak, Derya ;
Farahnik, Benjamin ;
Abrouk, Michael ;
Nakamura, Mio ;
Zhu, Tian Hao ;
Bhutani, Tina ;
Wei, Maria ;
Liao, Wilson .
AUTOIMMUNITY REVIEWS, 2016, 15 (04) :397-404
[5]
Epigallocatechin-3-gallate (EGCG) Suppresses the Trafficking of Lymphocytes to Epidermal Melanocytes via Inhibition of JAK2: Its Implication for Vitiligo Treatment [J].
Ning, Weixuan ;
Wang, Suiquan ;
Dong, Xiaowu ;
Liu, Dongyin ;
Fu, Lifang ;
Ha, Rong ;
Xe, Aie .
BIOLOGICAL & PHARMACEUTICAL BULLETIN, 2015, 38 (11) :1700-1706
[6]
Tofacitinib Citrate for the Treatment of Vitiligo A Pathogenesis-Directed Therapy [J].
Craiglow, Brittany G. ;
King, Brett A. .
JAMA DERMATOLOGY, 2015, 151 (10) :1110-1112
[7]
Oxidized tyrosinase: A possible antigenic stimulus for non-segmental vitiligo autoantibodies [J].
Al-Shobaili, Hani A. ;
Rasheed, Zafar .
JOURNAL OF DERMATOLOGICAL SCIENCE, 2015, 79 (03) :203-213
[8]
Tumour necrosis factor-α inhibition can stabilize disease in progressive vitiligo [J].
Webb, K. C. ;
Tung, R. ;
Winterfield, L. S. ;
Gottlieb, A. B. ;
Eby, J. M. ;
Henning, S. W. ;
Le Poole, I. C. .
BRITISH JOURNAL OF DERMATOLOGY, 2015, 173 (03) :641-650
[9]
Increased circulating Th17 cells and elevated serum levels of TGF-beta and IL-21 are correlated with human non-segmental vitiligo development [J].
Zhou, Li ;
Shi, Yu-Ling ;
Li, Kai ;
Hamzavi, Iltefat ;
Gao, Tian-Wen ;
Huggins, Richard H. ;
Lim, Henry W. ;
Mi, Qing-Sheng .
PIGMENT CELL & MELANOMA RESEARCH, 2015, 28 (03) :324-329
[10]
Regulatory T cells from active non-segmental vitiligo exhibit lower suppressive ability on CD8+ CLA+ T cells [J].
Lin, Mao ;
Zhang, Bao-Xiang ;
Shen, Nan ;
Dong, Xue-Jiao ;
Zhang, Ci ;
Qi, Xiao-Yi ;
Zhu, Jie ;
Li, Yu-Zhong ;
Man, Mao-Qiang ;
Tu, Cai-Xia .
EUROPEAN JOURNAL OF DERMATOLOGY, 2014, 24 (06) :676-682