HPV在宫颈癌发病中的作用机制研究

[11] Transcription independent triggering of the extrinsic pathway of apoptosis by human papillomavirus 18 E2 protein. Demeret C,Garcia Carranca A,Thierry F. Oncegene . 2003

[12] A prospective study showing long-term infection with human papillomavirus 16 before the development of cervical carcinoma in situ. Ylitalo N,Josefsson A,Melbye M,et al. Cancer Research . 2000

[13] The human papillomavirus type 16 E6 gene alone is sufficient to induce carcinomas in transgenic animals. Song S,Pitot HC,Lambert PF. Journal of Virology . 1999

[14] P53 immunohistochemical expression: messages in cervical Carcinogenesis. Cheah PL,Looi LM. The Journal of Pathology . 2002

[15] Induction of apoptosis in cervical carcinoma cells by peptide aptamers that bind to the HPV-16 E7 oncoprotein. Nauenburg S,Zwerschke W,Jansen Durr P. The FASEB Journal . 2001

[16] Association between activated K-ras and c-erbB-2 oncogenes with "high-risk" and "low-risk" human papilloma virus types in preinvasive cervical lesions. Mouron SA,Abba MC,Guerci A, et al. Mutation Research . 2000

[17] Both E6 and E7 oncoproteins of human papillomavirus 16 inhibit IL-18-induced IFN gamma production in human peripheral blood mononueclear and NK cells. Lee SJ,Cho YS,Cho MC,et al. J Immunol . 2001

[18] Interleukin-2in-hibits proliferation of HPV-associated tumor cells and halts tu-mor growth in vivo. CASANA PH,HERNANDEZ H,ARANA MJ. Biochemical and Biophysical Research Communications . 2002

[19] Relationship between steroid hormone contraceptives and HPV, cervical intraepithelial neoplasia and cervical carcinoma. de Villiers EM. International Journal of Cancer . 2003

[20] Herpes simplex virus-2 as a human papillomavirus cofactor in the etiology of invasive cervical cancer. Smith JS,Herrero R,Bosetti C,et al. Journal of the National Cancer Institute . 2002

[11] Transcription independent triggering of the extrinsic pathway of apoptosis by human papillomavirus 18 E2 protein. Demeret C,Garcia Carranca A,Thierry F. Oncegene . 2003

[12] A prospective study showing long-term infection with human papillomavirus 16 before the development of cervical carcinoma in situ. Ylitalo N,Josefsson A,Melbye M,et al. Cancer Research . 2000

[13] The human papillomavirus type 16 E6 gene alone is sufficient to induce carcinomas in transgenic animals. Song S,Pitot HC,Lambert PF. Journal of Virology . 1999

[14] P53 immunohistochemical expression: messages in cervical Carcinogenesis. Cheah PL,Looi LM. The Journal of Pathology . 2002

[15] Induction of apoptosis in cervical carcinoma cells by peptide aptamers that bind to the HPV-16 E7 oncoprotein. Nauenburg S,Zwerschke W,Jansen Durr P. The FASEB Journal . 2001

[16] Association between activated K-ras and c-erbB-2 oncogenes with "high-risk" and "low-risk" human papilloma virus types in preinvasive cervical lesions. Mouron SA,Abba MC,Guerci A, et al. Mutation Research . 2000

[17] Both E6 and E7 oncoproteins of human papillomavirus 16 inhibit IL-18-induced IFN gamma production in human peripheral blood mononueclear and NK cells. Lee SJ,Cho YS,Cho MC,et al. J Immunol . 2001

[18] Interleukin-2in-hibits proliferation of HPV-associated tumor cells and halts tu-mor growth in vivo. CASANA PH,HERNANDEZ H,ARANA MJ. Biochemical and Biophysical Research Communications . 2002

[19] Relationship between steroid hormone contraceptives and HPV, cervical intraepithelial neoplasia and cervical carcinoma. de Villiers EM. International Journal of Cancer . 2003

[20] Herpes simplex virus-2 as a human papillomavirus cofactor in the etiology of invasive cervical cancer. Smith JS,Herrero R,Bosetti C,et al. Journal of the National Cancer Institute . 2002