代谢综合征发病的炎症反应机制研究进展

被引：7

作者：

许梅花 ^{[1
]}

熊英环 ^{[2
]}

方今女 ^{[2
]}

机构：

[1] 延边大学护理学院

[2] 延边大学医学院预防医学教研部

来源：

延边大学医学学报 | 2013年 / 36卷 / 02期

关键词：

炎症反应; 代谢综合征; 胰岛素抵抗;

D O I：

10.16068/j.1000-1824.2013.02.013

中图分类号：

R589 [代谢病];

学科分类号：

100201 [内科学];

摘要：

<正>众所周知,高血压、血脂异常、糖尿病、肥胖等不仅是心血管疾病的危险因素,同时也是成人常见的代谢异常病.20世纪以来,人们开始观察到这些常见的代谢异常病常同时出现在同一个体,即多个代谢异常的个体聚集现象,且称为代谢综合征

引用

页码：150 / 153

页数：4

共 22 条

[1]

High-molecular-weight adiponectin is a predictor of progression to metabolic syndrome: a population-based 6-year follow-up study in Japanese men [J].

Seino, Yoshie ;

Hirose, Hiroshi ;

Saito, Ikuo ;

Itoh, Hiroshi .

METABOLISM-CLINICAL AND EXPERIMENTAL, 2009, 58 (03) :355-360

[2]

Adiponectin, cardiovascular function, and hypertension [J].

Wang, Zhao V. ;

Scherer, Philipp E. .

HYPERTENSION, 2008, 51 (01) :8-14

[3]

Inflammation, obesity, and the metabolic syndrome [J].

Yudkin, J. S. .

HORMONE AND METABOLIC RESEARCH, 2007, 39 (10) :707-709

[4]

The Metabolic Syndrome: Inflammation; Diabetes Mellitus; and Cardiovascular Disease.[J].Steven M. Haffner.The American Journal of Cardiology.2006, 2

[5]

The metabolic syndrome - a new worldwide definition [J].

Alberti, KGMM ;

Zimmet, P ;

Shaw, J .

LANCET, 2005, 366 (9491) :1059-1062

[6]

METABOLIC SYNDROME: A Clinical and Molecular Perspective.[J].David E. Moller;Keith D. Kaufman.Annual Review of Medicine.2005, 1

[7]

Leptin: linking adipocyte metabolism with cardiovascular and autoimmune diseases [J].

Peelman, F ;

Waelput, W ;

Iserentant, H ;

Lavens, D ;

Eyckerman, S ;

Zabeau, L ;

Tavernier, J .

PROGRESS IN LIPID RESEARCH, 2004, 43 (04) :283-301

[8]

Insulin resistance in adipose tissue:: direct and indirect effects of tumor necrosis factor-α [J].

Ruan, H ;

Lodish, HF .

CYTOKINE & GROWTH FACTOR REVIEWS, 2003, 14 (05) :447-455

[9]

The adipocyte in insulin resistance: key molecules and the impact of the thiazolidinediones [J].

Arner, P .

TRENDS IN ENDOCRINOLOGY AND METABOLISM, 2003, 14 (03) :137-145

[10]

Interleukin (IL)-6 mRNA expression is stimulated by insulin, isoproterenol, tumour necrosis factor alpha, growth hormone, and IL-6 in 3T3-L1 adipocytes [J].

Fasshauer, M ;

Klein, J ;

Lossner, U ;

Paschke, R .

HORMONE AND METABOLIC RESEARCH, 2003, 35 (03) :147-152

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