脑缺血-再灌注损伤后的炎性反应过程及机制的研究进展

被引：26

作者：

张迪 ^{[1
]}

任长虹 ^{[2
]}

吉训明 ^{[2
]}

朱榆红 ^{[1
]}

机构：

[1] 昆明医科大学第二附属医院神经内科

[2] 首都医科大学宣武医院低氧研究所

来源：

中国脑血管病杂志 | 2013年 / 07期

关键词：

脑缺血; 再灌注损伤; 炎性反应; 免疫;

D O I：

暂无

中图分类号：

R743 [脑血管疾病];

学科分类号：

摘要：

<正>脑缺血诱导的再灌注损伤是由多种机制参与的一种复杂的病理生理学过程,主要包括自由基过度形成、兴奋性氨基酸毒性作用、细胞内钙超载和炎性反应等多种机制。这些因素之间又互相影响,进一步促进脑缺血-再灌注损伤后的神经功能破坏。炎性反应在脑缺血-再灌注损伤中起着关键性

引用

页码：383 / 387

页数：5

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