DNA methylation and methyl-CpG binding proteins: developmental requirements and function

被引:324
作者
Bogdanovic, Ozren [1 ]
Veenstra, Gert Jan C. [1 ]
机构
[1] Radboud Univ Nijmegen, Dept Mol Biol, Fac Sci, Nijmegen Ctr Mol Life Sci, NL-6525 ED Nijmegen, Netherlands
关键词
DEPENDENT TRANSCRIPTIONAL REPRESSOR; CHROMATIN-REMODELING COMPLEX; EMBRYONIC STEM-CELLS; DE-NOVO METHYLATION; HISTONE DEACETYLASE COMPLEX; HONEYBEE APIS-MELLIFERA; RETT-SYNDROME; HUMAN CANCER; MAINTENANCE METHYLATION; MAMMALIAN DEVELOPMENT;
D O I
10.1007/s00412-009-0221-9
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
DNA methylation is a major epigenetic modification in the genomes of higher eukaryotes. In vertebrates, DNA methylation occurs predominantly on the CpG dinucleotide, and approximately 60% to 90% of these dinucleotides are modified. Distinct DNA methylation patterns, which can vary between different tissues and developmental stages, exist on specific loci. Sites of DNA methylation are occupied by various proteins, including methyl-CpG binding domain (MBD) proteins which recruit the enzymatic machinery to establish silent chromatin. Mutations in the MBD family member MeCP2 are the cause of Rett syndrome, a severe neurodevelopmental disorder, whereas other MBDs are known to bind sites of hypermethylation in human cancer cell lines. Here, we review the advances in our understanding of the function of DNA methylation, DNA methyltransferases, and methyl-CpG binding proteins in vertebrate embryonic development. MBDs function in transcriptional repression and long-range interactions in chromatin and also appear to play a role in genomic stability, neural signaling, and transcriptional activation. DNA methylation makes an essential and versatile epigenetic contribution to genome integrity and function.
引用
收藏
页码:549 / 565
页数:17
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