Commentary on "Alpha-synuclein and mitochondria: a tangled skein"

[1] Chaperone suppression of α-synuclein toxicity in a Drosophila model for Parkinson's disease [J]. SCIENCE, 2002, 295 (5556) : 865 - 868

[2] Chronic systemic pesticide exposure reproduces features of Parkinson's disease [J]. NATURE NEUROSCIENCE, 2000, 3 (12) : 1301 - 1306

[3] Bouet V, 2007, EXP NEUROL, V203, P555

[4] Resistance of α-synuclein null mice to the parkinsonian neurotoxin MPTP [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2002, 99 (22) : 14524 - 14529

[5] A Drosophila model of Parkinson's disease [J]. NATURE, 2000, 404 (6776) : 394 - 398

[6] In situ and in vitro study of colocalization and segregation of α-synuclein, ubiquitin, and lipids in Lewy bodies [J]. EXPERIMENTAL NEUROLOGY, 2000, 166 (02) : 324 - 333

[7] Gene expression profile of oxidant stress and neurodegeneration in transgenic mice deficient in α-tocopherol transfer protein [J]. FREE RADICAL BIOLOGY AND MEDICINE, 2003, 35 (11) : 1343 - 1354

[8] Golgi fragmentation occurs in the cells with prefibrillar α-synuclein aggregates and precedes the formation of fibrillar inclusion [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2002, 277 (50) : 48984 - 48992

[9] Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2003, 100 (07) : 4078 - 4083

[10] α-synuclein promotes mitochondrial deficit and oxidative stress [J]. AMERICAN JOURNAL OF PATHOLOGY, 2000, 157 (02) : 401 - 410

[1] Chaperone suppression of α-synuclein toxicity in a Drosophila model for Parkinson's disease [J]. SCIENCE, 2002, 295 (5556) : 865 - 868

[2] Chronic systemic pesticide exposure reproduces features of Parkinson's disease [J]. NATURE NEUROSCIENCE, 2000, 3 (12) : 1301 - 1306

[3] Bouet V, 2007, EXP NEUROL, V203, P555

[4] Resistance of α-synuclein null mice to the parkinsonian neurotoxin MPTP [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2002, 99 (22) : 14524 - 14529

[5] A Drosophila model of Parkinson's disease [J]. NATURE, 2000, 404 (6776) : 394 - 398

[6] In situ and in vitro study of colocalization and segregation of α-synuclein, ubiquitin, and lipids in Lewy bodies [J]. EXPERIMENTAL NEUROLOGY, 2000, 166 (02) : 324 - 333

[7] Gene expression profile of oxidant stress and neurodegeneration in transgenic mice deficient in α-tocopherol transfer protein [J]. FREE RADICAL BIOLOGY AND MEDICINE, 2003, 35 (11) : 1343 - 1354

[8] Golgi fragmentation occurs in the cells with prefibrillar α-synuclein aggregates and precedes the formation of fibrillar inclusion [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2002, 277 (50) : 48984 - 48992

[9] Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2003, 100 (07) : 4078 - 4083

[10] α-synuclein promotes mitochondrial deficit and oxidative stress [J]. AMERICAN JOURNAL OF PATHOLOGY, 2000, 157 (02) : 401 - 410