Five Questions on Prion Diseases

被引:22
作者
Aguzzi, Adriano [1 ]
Zhu, Caihong [1 ]
机构
[1] Univ Zurich Hosp, Inst Neuropathol, CH-8091 Zurich, Switzerland
基金
瑞士国家科学基金会; 欧洲研究理事会;
关键词
FOLLICULAR DENDRITIC CELLS; AMYLOID-BETA OLIGOMERS; VARIANT CJD; PROTEIN; MICE; REPLICATION; SCRAPIE; IMPAIRMENT; TOXICITY; NEUROINVASION;
D O I
10.1371/journal.ppat.1002651
中图分类号
Q93 [微生物学];
学科分类号
071005 [微生物学];
摘要
[No abstract available]
引用
收藏
页数:4
相关论文
共 29 条
[1]
Prion research: the next frontiers [J].
Aguzzi, A ;
Weissmann, C .
NATURE, 1997, 389 (6653) :795-798
[2]
Synthetic amyloid-β oligomers impair long-term memory independently of cellular prion protein [J].
Balducci, Claudia ;
Beeg, Marten ;
Stravalaci, Matteo ;
Bastone, Antonio ;
Sclip, Alessandra ;
Biasini, Emiliano ;
Tapella, Laura ;
Colombo, Laura ;
Manzoni, Claudia ;
Borsello, Tiziana ;
Chiesa, Roberto ;
Gobbi, Marco ;
Salmona, Mario ;
Forloni, Gianluigi .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2010, 107 (05) :2295-2300
[3]
Lethal recessive myelin toxicity of prion protein lacking its central domain [J].
Baumann, Frank ;
Tolnay, Markus ;
Brabeck, Christine ;
Pahnke, Jens ;
Kloz, Ulrich ;
Niemann, Hartmut H. ;
Heikenwalder, Mathias ;
Ruelicke, Thomas ;
Buerkle, Alexander ;
Aguzzi, Adriano .
EMBO JOURNAL, 2007, 26 (02) :538-547
[4]
Small is not beautiful:: antagonizing functions for the prion protein PrPC and its homologue Dpl [J].
Behrens, A ;
Aguzzi, A .
TRENDS IN NEUROSCIENCES, 2002, 25 (03) :150-154
[5]
BERNOULLI C, 1977, LANCET, V1, P478
[6]
Normal host prion protein necessary for scrapie-induced neurotoxicity [J].
Brandner, S ;
Isenmann, S ;
Raeber, A ;
Fischer, M ;
Sailer, A ;
Kobayashi, Y ;
Marino, S ;
Weissmann, C ;
Aguzzi, A .
NATURE, 1996, 379 (6563) :339-343
[7]
Scrapie replication in lymphoid tissues depends on prion protein-expressing follicular dendritic cells [J].
Brown, KL ;
Stewart, K ;
Ritchie, DL ;
Mabbott, NA ;
Williams, A ;
Fraser, H ;
Morrison, WI ;
Bruce, ME .
NATURE MEDICINE, 1999, 5 (11) :1308-1312
[8]
Transmissions to mice indicate that 'new variant' CJD is caused by the BSE agent [J].
Bruce, ME ;
Will, RG ;
Ironside, JW ;
McConnell, I ;
Drummond, D ;
Suttie, A ;
McCardle, L ;
Chree, A ;
Hope, J ;
Birkett, C ;
Cousens, S ;
Fraser, H ;
Bostock, CJ .
NATURE, 1997, 389 (6650) :498-501
[9]
Prion protein and Aβ-related synaptic toxicity impairment [J].
Calella, Anna Maria ;
Farinelli, Melissa ;
Nuvolone, Mario ;
Mirante, Osvaldo ;
Moos, Rita ;
Falsig, Jeppe ;
Mansuy, Isabelle M. ;
Aguzzi, Adriano .
EMBO MOLECULAR MEDICINE, 2010, 2 (08) :306-314
[10]
Neurological illness in transgenic mice expressing a prion protein with an insertional mutation [J].
Chiesa, R ;
Piccardo, P ;
Ghetti, B ;
Harris, DA .
NEURON, 1998, 21 (06) :1339-1351