Pathogenesis of alcoholic liver disease-recent advances

被引：27

作者：

Nanji, AA

Su, GL

Laposata, M

French, SW

机构：

[1] Queen Mary Hosp, Clin Biochem Unit, Hong Kong, Hong Kong, Peoples R China

[2] Harbor UCLA Med Ctr, Dept Pathol, Torrance, CA 90509 USA

[3] Harvard Univ, Sch Med, Boston, MA USA

[4] Massachusetts Gen Hosp, Dept Pathol, Div Lab Med, Boston, MA 02114 USA

[5] Univ Michigan, Med Ctr, Dept Med, Ann Arbor, MI 48109 USA

[6] Univ Hong Kong, Dept Pathol, Hong Kong, Hong Kong, Peoples R China

[7] Univ Hong Kong, Ctr Study Liver Dis, Hong Kong, Hong Kong, Peoples R China

来源：

ALCOHOLISM-CLINICAL AND EXPERIMENTAL RESEARCH | 2002年 / 26卷 / 05期

关键词：

alcoholic liver disease; cyclooxygenase; lipopolysaccharide; fatty acid esters; oxidative stress;

D O I：

10.1111/j.1530-0277.2002.tb02598.x

中图分类号：

R194 [卫生标准、卫生检查、医药管理];

学科分类号：

摘要：

The article summarizes the proceedings of a symposium on recent advances in research on the pathogenesis of alcoholic liver disease at the 2001 RSA meeting in Montreal, Canada. The chairs were Amin A. Nanji and Samuel W. French. The presentations were (1) Role of inflammatory mediators in alcoholic liver injury by Amin A. Nanji, (2) Role of endotoxin, lipopolysaccharide binding protein, CD14 and Toll receptors in alcoholic liver injury by Grace Su, (3) Fatty acid ethyl esters: toxicity, metabolism and markers of ethanol intake by Michael Laposata, and (4) Cyclic changes in gene expression when rats are fed alcohol at a constant rate by Samuel W. French.

引用

页码：731 / 736

页数：6

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