Adeno-associated virus vector-mediated IL-10 gene delivery prevents type 1 diabetes in NOD mice

被引:113
作者
Goudy, K
Song, SH
Wasserfall, C
Zhang, YC
Kapturczak, M
Muir, A
Powers, M
Scott-Jorgensen, M
Campbell-Thompson, M
Crawford, JM
Ellis, TM
Flotte, TR
Atkinson, MA
机构
[1] Univ Florida, Coll Med, Dept Pathol, Gainesville, FL 32610 USA
[2] Univ Florida, Genet Inst, Gainesville, FL 32610 USA
[3] Univ Florida, Powell Gene Therapy Ctr, Gainesville, FL 32610 USA
[4] Univ Florida, Dept Pediat, Gainesville, FL 32610 USA
[5] Univ Florida, Dept Med, Gainesville, FL 32610 USA
关键词
D O I
10.1073/pnas.251532298
中图分类号
O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];
学科分类号
07 ; 0710 ; 09 ;
摘要
The development of spontaneous autoimmune diabetes in nonobese diabetic (NOD) mice provides for their use as a model of human type 1 diabetes. To test the feasibility of muscle-directed gene therapy to prevent type 1 diabetes, we developed recombinant adeno-associated virus (rAAV) vectors containing murine cDNAs for immunomodulatory cytokines IL-4 or IL-10. Skeletal muscle transduction of female NOD mice with IL-10, but not IL-4, completely abrogated diabetes. rAAV-IL-10 transduction attenuated the production of insulin autoantibodies, quantitatively reduced pancreatic insulitis, maintained islet insulin content, and altered splenocyte cytokine responses to mitogenic stimulation. The beneficial effects were host specific, as adoptive transfer of splenocytes from rAAV IL-10-treated animals rapidly imparted diabetes in naive hosts, and the cells contained no protective immunomodulatory capacity, as defined through adoptive cotransfer analyses. These results indicate the utility for rAAV, a vector with advantages for therapeutic gene delivery, to transfer immunoregulatory cytokines capable of preventing type 1 diabetes. In addition, these studies provide foundational support for the concept of using immunoregulatory agents delivered by rAAV to modulate a variety of disorders associated with deleterious immune responses, including allergic reactions, transplantation rejection, immunodeficiencies, and autoimmune disorders.
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页码:13913 / 13918
页数:6
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