Wounding smad

被引：30

作者：

Massagué, J ^{[1
]}

机构：

[1] Mem Sloan Kettering Canc Ctr, Howard Hughes Med Inst, Cell Biol Program, New York, NY 10021 USA

来源：

NATURE CELL BIOLOGY | 1999年 / 1卷 / 05期

关键词：

D O I：

10.1038/12944

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

TGF-beta is a cytokine that enhances wound healing, a process involving diverse TGF-beta-responsive cell types. Mice with a deletion of the Smad3 gene exhibit an Increased rate of wound healing. The lack of Smad3 seems to modify the cellular response to TGF-beta and may suggest an intriguing molecular basis to this process.

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收藏

页码：E117 / E119

页数：3

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