Going mad with Smads

被引：28

作者：

Zhou, SB ^{[1
]}

Kinzler, KW ^{[1
]}

Vogelstein, B ^{[1
]}

机构：

[1] Johns Hopkins Oncol Ctr, Baltimore, MD 21231 USA

来源：

NEW ENGLAND JOURNAL OF MEDICINE | 1999年 / 341卷 / 15期

关键词：

D O I：

10.1056/NEJM199910073411510

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

[No abstract available]

引用

页码：1144 / 1146

页数：3

共 8 条

[1]

Datto MB, 1999, MOL CELL BIOL, V19, P2495

[2]

Grady WM, 1999, CANCER RES, V59, P320

[3] TGF-β signal transduction [J].

Massagué, J .

ANNUAL REVIEW OF BIOCHEMISTRY, 1998, 67 :753-791

[4]

Riggins GJ, 1997, CANCER RES, V57, P2578

[5] Intestinal tumorigenesis in compound mutant mice of both Dpc4 (Smad4) and Apc genes [J].

Takaku, K ;

Oshima, M ;

Miyoshi, H ;

Matsui, M ;

Seldin, MF ;

Taketo, MM .

CELL, 1998, 92 (05) :645-656

[6]

WEINSTEIN IB, 1996, FUNDAMENTALS CANC PR, P76

[7] Targeted disruption of SMAD3 results in impaired mucosal immunity and diminished T cell responsiveness to TGF-β [J].

Yang, X ;

Letterio, JJ ;

Lechleider, RJ ;

Chen, L ;

Hayman, R ;

Gu, H ;

Roberts, AB ;

Deng, CX .

EMBO JOURNAL, 1999, 18 (05) :1280-1291

[8] Smad3 mutant mice develop metastatic colorectal cancer [J].

Zhu, YA ;

Richardson, JA ;

Parada, LF ;

Graff, JM .

CELL, 1998, 94 (06) :703-714

← 1 →