p53 signaling and cell cycle checkpoints

被引：156

作者：

Stewart, ZA

Pietenpol, JA

机构：

[1] Vanderbilt Univ, Sch Med, Dept Biochem, Ctr Mol Toxicol, Nashville, TN 37232 USA

[2] Vanderbilt Univ, Sch Med, Vanderbilt Ingram Canc Ctr, Nashville, TN 37232 USA

来源：

CHEMICAL RESEARCH IN TOXICOLOGY | 2001年 / 14卷 / 03期

关键词：

D O I：

10.1021/tx000199t

中图分类号：

R914 [药物化学];

学科分类号：

100701 ;

摘要：

[No abstract available]

引用

页码：243 / 263

页数：21

共 328 条

[11] Tumor suppressor p53 is required to modulate BRCA1 expression [J].

Arizti, P ;

Fang, L ;

Park, I ;

Yin, YX ;

Solomon, E ;

Ouchi, T ;

Aaronson, SA ;

Lee, SW .

MOLECULAR AND CELLULAR BIOLOGY, 2000, 20 (20) :7450-7459

[12]

Ashcroft M, 1999, MOL CELL BIOL, V19, P1751

[13] The role of p53 in tumour suppression: lessons from mouse models [J].

Attardi, LD ;

Jacks, T .

CELLULAR AND MOLECULAR LIFE SCIENCES, 1999, 55 (01) :48-63

[14] Recruitment of p300/CBP in p53-dependent signal pathways [J].

Avantaggiati, ML ;

Ogryzko, V ;

Gardner, K ;

Giordano, A ;

Levine, AS ;

Kelly, K .

CELL, 1997, 89 (07) :1175-1184

[15]

Badie C, 2000, BRIT J CANCER, V82, P642

[16] SUPPRESSION OF HUMAN COLORECTAL-CARCINOMA CELL-GROWTH BY WILD-TYPE-P53 [J].

BAKER, SJ ;

MARKOWITZ, S ;

FEARON, ER ;

WILLSON, JKV ;

VOGELSTEIN, B .

SCIENCE, 1990, 249 (4971) :912-915

[17] CHROMOSOME-17 DELETIONS AND P53 GENE-MUTATIONS IN COLORECTAL CARCINOMAS [J].

BAKER, SJ ;

FEARON, ER ;

NIGRO, JM ;

HAMILTON, SR ;

PREISINGER, AC ;

JESSUP, JM ;

VANTUINEN, P ;

LEDBETTER, DH ;

BARKER, DF ;

NAKAMURA, Y ;

WHITE, R ;

VOGELSTEIN, B .

SCIENCE, 1989, 244 (4901) :217-221

[18] Enhanced phosphorylation of p53 by ATN in response to DNA damage [J].

Banin, S ;

Moyal, L ;

Shieh, SY ;

Taya, Y ;

Anderson, CW ;

Chessa, L ;

Smorodinsky, NI ;

Prives, C ;

Reiss, Y ;

Shiloh, Y ;

Ziv, Y .

SCIENCE, 1998, 281 (5383) :1674-1677

[19] MDM2 EXPRESSION IS INDUCED BY WILD TYPE-P53 ACTIVITY [J].

BARAK, Y ;

JUVEN, T ;

HAFFNER, R ;

OREN, M .

EMBO JOURNAL, 1993, 12 (02) :461-468

[20] A PROTEOLYTIC FRAGMENT FROM THE CENTRAL REGION OF P53 HAS MARKED SEQUENCE-SPECIFIC DNA-BINDING ACTIVITY WHEN GENERATED FROM WILD-TYPE BUT NOT FROM ONCOGENIC MUTANT P53-PROTEIN [J].

BARGONETTI, J ;

MANFREDI, JJ ;

CHEN, XB ;

MARSHAK, DR ;

PRIVES, C .

GENES & DEVELOPMENT, 1993, 7 (12B) :2565-2574

← 1 2 3 4 5 6 7 8 9 10 →