The tolerogenic molecule HLA-G

被引：59

作者：

Carosella, Edgardo D. ^{[1
]}

机构：

[1] Univ Paris 07, UMR E, Serv Rech Hematoimmunol, CEA I2BM,IUH,Hop St Louis, F-75010 Paris, France

来源：

IMMUNOLOGY LETTERS | 2011年 / 138卷 / 01期

关键词：

ANTIGEN-PRESENTING CELLS; G EXPRESSION; T-CELLS; TUMOR-CELLS; NK CELLS; ESCAPE; INHIBITION; ACCEPTANCE; CANCER;

D O I：

10.1016/j.imlet.2011.02.011

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

071005 [微生物学]; 100108 [医学免疫学];

摘要：

[No abstract available]

引用

页码：22 / 24

页数：3

共 21 条

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[2]

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ILT2/HLA-G interaction impairs NK-cell functions through the inhibition of the late but not the early events of the NK-cell activating synapse [J].

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HLA-G up-regulates ILT2, ILT3, ILT4, and KIR2DL4 in antigen presenting cells, NK cells, and T cells [J].

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HLA-G1-expressing antigen-presenting cells induce immunosuppressive CD4+ T cells [J].

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Immune regulation by pretenders: cell-to-cell transfers of HLA-G make effector T cells act as regulatory cells [J].

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Implication of HLA-G molecule in heart-graft acceptance [J].

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