Oxidant stress as a marker for cardiovascular events - Ox marks the spot

被引：103

作者：

Landmesser, U

Harrison, DG

机构：

[1] Emory Univ, Sch Med, Div Cardiol, Dept Med, Atlanta, GA 30322 USA

[2] Atlanta Vet Adm Hosp, Atlanta, GA USA

来源：

CIRCULATION | 2001年 / 104卷 / 22期

关键词：

editorials; endothelium; oxidant stress; coronary disease; nitric oxide;

D O I：

10.1161/circ.104.22.2638

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：2638 / 2640

页数：3

共 24 条

[21] Improvement of nitric oxide-dependent vasodilatation by HMG-CoA reductase inhibitors through attenuation of endothelial superoxide anion formation [J].

Wagner, AH ;

Köhler, T ;

Rückschloss, U ;

Just, I ;

Hecker, M .

ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2000, 20 (01) :61-69

[22] Increased NADH-oxidase-mediated superoxide production in the early stages of atherosclerosis -: Evidence for involvement of the renin-angiotensin system [J].

Warnholtz, A ;

Nickenig, G ;

Schulz, E ;

Macharzina, R ;

Bräsen, JH ;

Skatchkov, M ;

Heitzer, T ;

Stasch, JP ;

Griendling, KK ;

Harrison, DG ;

Böhm, M ;

Meinertz, T ;

Münzel, T .

CIRCULATION, 1999, 99 (15) :2027-2033

[23] Inhibition of geranylgeranylation reduces angiotensin II-mediated free radical production in vascular smooth muscle cells:: Involvement of angiotensin AT1 receptor expression and Rac1 GTPase [J].

Wassmann, S ;

Laufs, U ;

Bäumer, AT ;

Müller, K ;

Konkol, C ;

Sauer, H ;

Böhm, M ;

Nickenig, G .

MOLECULAR PHARMACOLOGY, 2001, 59 (03) :646-654

[24]

Yusuf S, 2000, NEW ENGL J MED, V342, P154, DOI 10.1056/NEJM200001203420302

← 1 2 3 →