Psoriatic arthritis:: clinical aspects, genetics, and the role of T cells

被引：18

作者：

Höhler, T ^{[1
]}

Märker-Hermann, E ^{[1
]}

机构：

[1] Univ Klin Mainz, Med Klin & Poliklin 1, D-55101 Mainz, Germany

来源：

CURRENT OPINION IN RHEUMATOLOGY | 2001年 / 13卷 / 04期

关键词：

D O I：

10.1097/00002281-200107000-00005

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

In the last 2 years there has been considerable progress in investigating the genetic and immunologic background of psoriasis and psoriatic arthritis. This review focuses on genetics and the role of T-cells in the immunopathogenesis of the disease, with particular reference to psoriatic arthritis. (C) 2001 Lippincott Williams & Wilkins, Inc.

引用

页码：273 / 279

页数：7

共 70 条

[31] Genetics of psoriasis [J].

Henseler, T .

ARCHIVES OF DERMATOLOGICAL RESEARCH, 1998, 290 (09) :463-476

[32] A TNF-alpha promoter polymorphism is associated with juvenile onset psoriasis and psoriatic arthritis [J].

Hohler, T ;

Kruger, A ;

Schneider, PM ;

Schopf, RE ;

Knop, J ;

Rittner, C ;

zumBuschenfelde, KHM ;

MarkerHermann, E .

JOURNAL OF INVESTIGATIVE DERMATOLOGY, 1997, 109 (04) :562-565

[33]

Hohler T, 1996, CLIN EXP RHEUMATOL, V14, P661

[34] TAP-polymorphisms in juvenile onset psoriasis and psoriatic arthritis [J].

Hohler, T ;

Weinmann, A ;

Schneider, PM ;

Rittner, C ;

Schopf, RE ;

Knop, J ;

Hasenclever, P ;

zumBuschenfelde, KHM ;

MarkerHermann, E .

HUMAN IMMUNOLOGY, 1996, 51 (01) :49-54

[35] Corneodesmosin gene polymorphism demonstrates strong linkage disequilibrium with HLA and association with psoriasis vulgaris [J].

Jenisch, S ;

Koch, S ;

Henseler, T ;

Nair, RP ;

Elder, JT ;

Watts, CE ;

Westphal, E ;

Voorhees, JJ ;

Christophers, E ;

Krönke, M .

TISSUE ANTIGENS, 1999, 54 (05) :439-449

[36] Linkage disequilibrium analysis of familial psoriasis:: identification of multiple disease associated MHC haplotypes [J].

Jenisch, S ;

Westphal, E ;

Nair, RP ;

Stuart, P ;

Voorhees, JJ ;

Christophers, E ;

Krönke, M ;

Elder, JT ;

Henseler, T .

TISSUE ANTIGENS, 1999, 53 (02) :135-146

[37] Different transcriptional activity and in vitro TNF-α production in psoriasis patients carrying the TNF-α 238A promoter polymorphism [J].

Kaluza, W ;

Reuss, E ;

Grossmann, S ;

Hug, R ;

Schopf, RE ;

Galle, PR ;

Maerker-Hermann, E ;

Hoehler, T .

JOURNAL OF INVESTIGATIVE DERMATOLOGY, 2000, 114 (06) :1180-1183

[38] Subsets in psoriatic arthritis formed by cluster analysis [J].

Koó, É ;

Nagy, Z ;

Seszták, M ;

Ujfalussy, I ;

Merétey, K ;

Böhm, U ;

Forgács, S ;

Szilágy, M ;

Czirják, L ;

Farkas, V .

CLINICAL RHEUMATOLOGY, 2001, 20 (01) :36-43

[39] GENETIC DISSECTION OF COMPLEX TRAITS - GUIDELINES FOR INTERPRETING AND REPORTING LINKAGE RESULTS [J].

LANDER, E ;

KRUGLYAK, L .

NATURE GENETICS, 1995, 11 (03) :241-247

[40] The variable incidence of psoriasis in sub-Saharan Africa [J].

Leder, RO ;

Farber, EM .

INTERNATIONAL JOURNAL OF DERMATOLOGY, 1997, 36 (12) :911-919

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