Recent advances in the pathophysiology of ischemic acute renal failure

[101] PARK KM, 2003, IN PRESS J BIOL CHEM

[102] Injury in renal ischemia-reperfusion is independent from immunoglobulins and T lymphocytes [J]. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, 2002, 282 (02) : F352 - F357

[103] Etomoxir-induced PPARα-modulated enzymes protect during acute renal failure [J]. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, 2000, 278 (04) : F667 - F675

[104] ATP depletion of tubular cells causes dissociation of the zonula adherens and nuclear translocation of β-catenin and LEF-1 [J]. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY, 2002, 13 (05): : 1152 - 1161

[105] Pathophysiological role of T lymphocytes in renal ischemia-reperfusion injury in mice [J]. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, 2000, 279 (03) : F525 - F531

[106] TNF-α mediates chemokine and cytokine expression and renal injury in cisplatin nephrotoxicity [J]. JOURNAL OF CLINICAL INVESTIGATION, 2002, 110 (06) : 835 - 842

[107] ACTIVATION OF POTASSIUM CHANNELS CONTRIBUTES TO HYPOXIC INJURY IN PROXIMAL TUBULES [J]. JOURNAL OF CLINICAL INVESTIGATION, 1994, 94 (06) : 2289 - 2294

[108] RODRIGUES MR, 2002, BIOCHEM BIOPH RES CO, V869, P873

[109] Difficulties in understanding human "acute tubular necrosis": Limited data and flawed animal models [J]. KIDNEY INTERNATIONAL, 2001, 60 (04) : 1220 - 1224

[110] EXPRESSION OF CYTOKINE-LIKE GENES JE AND KC IS INCREASED DURING RENAL ISCHEMIA [J]. AMERICAN JOURNAL OF PHYSIOLOGY, 1991, 261 (06): : F1095 - F1101

[101] PARK KM, 2003, IN PRESS J BIOL CHEM

[102] Injury in renal ischemia-reperfusion is independent from immunoglobulins and T lymphocytes [J]. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, 2002, 282 (02) : F352 - F357

[103] Etomoxir-induced PPARα-modulated enzymes protect during acute renal failure [J]. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, 2000, 278 (04) : F667 - F675

[104] ATP depletion of tubular cells causes dissociation of the zonula adherens and nuclear translocation of β-catenin and LEF-1 [J]. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY, 2002, 13 (05): : 1152 - 1161

[105] Pathophysiological role of T lymphocytes in renal ischemia-reperfusion injury in mice [J]. AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY, 2000, 279 (03) : F525 - F531

[106] TNF-α mediates chemokine and cytokine expression and renal injury in cisplatin nephrotoxicity [J]. JOURNAL OF CLINICAL INVESTIGATION, 2002, 110 (06) : 835 - 842

[107] ACTIVATION OF POTASSIUM CHANNELS CONTRIBUTES TO HYPOXIC INJURY IN PROXIMAL TUBULES [J]. JOURNAL OF CLINICAL INVESTIGATION, 1994, 94 (06) : 2289 - 2294

[108] RODRIGUES MR, 2002, BIOCHEM BIOPH RES CO, V869, P873

[109] Difficulties in understanding human "acute tubular necrosis": Limited data and flawed animal models [J]. KIDNEY INTERNATIONAL, 2001, 60 (04) : 1220 - 1224

[110] EXPRESSION OF CYTOKINE-LIKE GENES JE AND KC IS INCREASED DURING RENAL ISCHEMIA [J]. AMERICAN JOURNAL OF PHYSIOLOGY, 1991, 261 (06): : F1095 - F1101