Current progress in keloid research and treatment

被引：218

作者：

Butler, Paris D. ^{[1
,3
]}

Longaker, Michael T. ^{[1
]}

Yang, George P. ^{[1
,2
]}

机构：

[1] Stanford Univ, Dept Surg, Sch Med, Stanford, CA 94305 USA

[2] Palo Alto VA Hlth Care Syst, Palo Alto, CA USA

[3] Univ Virginia, Sch Med, Dept Surg, Charlottesville, VA 22908 USA

来源：

JOURNAL OF THE AMERICAN COLLEGE OF SURGEONS | 2008年 / 206卷 / 04期

关键词：

D O I：

10.1016/j.jamcollsurg.2007.12.001

中图分类号：

R61 [外科手术学];

学科分类号：

摘要：

[No abstract available]

引用

页码：731 / 741

页数：11

共 94 条

[91] Increased CCN2 transcription in keloid fibroblasts requires cooperativity between AP-1 and SMAD binding sites [J].

Xia, Wei ;

Kong, Wuyi ;

Wang, Zhen ;

Phan, Toan-Thang ;

Lim, Ivor J. ;

Longaker, Michael T. ;

Yang, George P. .

ANNALS OF SURGERY, 2007, 246 (05) :886-895

[92] Differential transcriptional responses of keloid and normal keratinocytes to serum stimulation [J].

Xia, Wei ;

Phan, Toan-Thang ;

Lim, Ivor J. ;

Longaker, Michael T. ;

Yang, George P. .

JOURNAL OF SURGICAL RESEARCH, 2006, 135 (01) :156-163

[93] Overexpression of insulin-like growth factor-1 (IGF-I) receptor and the invasiveness of cultured keloid fibroblasts [J].

Yoshimoto, H ;

Ishihara, H ;

Ohtsuru, A ;

Akino, K ;

Murakami, R ;

Kuroda, H ;

Namba, H ;

Ito, M ;

Fujii, T ;

Yamashita, S .

AMERICAN JOURNAL OF PATHOLOGY, 1999, 154 (03) :883-889

[94]

ZHAHG Q, 2003, J INVEST DERMATOL, V121, P1005

← 1 2 3 4 5 6 7 8 9 10 →