Discoidin domain of Del1 protein contributes to its deposition in the extracellular matrix

被引:48
作者
Hidai, Chiaki
Kawana, Masatoshi
Kitano, Hisataka
Kokubun, Shinichiro
机构
[1] Nihon Univ, Sch Med, Dept Physiol, Itabashi Ku, Tokyo 1738610, Japan
[2] Nihon Univ, Sch Med, Res Ctr Adv Med, Tokyo 1738610, Japan
[3] Nihon Univ, Sch Med, Dept Dent Surg, Tokyo 1738610, Japan
[4] Tokyo Womens Med Univ, Aoyama Hosp, Tokyo 1070061, Japan
关键词
extracellular matrix; Del1; discoidin; epidermal growth factor; Cos-7; cells; mouse;
D O I
10.1007/s00441-007-0456-9
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
The extracellular matrix (ECM) acts as a critical factor during morphogenesis. Because the organization of the ECM directly influences the structure of tissues and organs, a determination of the way that ECM organization is regulated should help to clarify morphogenesis. We have analyzed the assembly of Del1, an ECM protein produced by endothelial cells in embryos, in the ECM. Del1 consists of three epidermal growth factor repeats (E1-E3) at its N-terminus and two discoidin domains (C1, C2) at its C-terminus. Experiments with various deletion mutants of Del1 have revealed that fragments containing the C-terminus of C1, which has a lectin-like structure, direct deposition in the ECM. The efficiency of deposition varies according to the presence of other domains in Del1. A fragment containing E3 and C1 has the strongest deposition activity, whereas fragments containing C2, which is highly homologous to C1, have low deposition activity. Digestion of ECM with hyaluronidase from bovine testis releases Del1 from the ECM, suggesting that glycosaminoglycans are involved in the deposition of Del1. In vivo gene transfer experiments have shown that fusion with the deposition domain of Del1 dramatically alters the distribution of exogenous proteins in mice. Thus, the extent of Del1 deposition may modify the organization of the ECM.
引用
收藏
页码:83 / 95
页数:13
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