The Genetics of Parkinson Disease

被引:215
作者
Bekris, Lynn M. [1 ,2 ]
Mata, Ignacio F. [1 ,3 ]
Zabetian, Cyrus P. [1 ,3 ]
机构
[1] Vet Affairs Puget Sound Hlth Care Syst, Geriatr Res Educ & Clin Ctr, Seattle, WA 98108 USA
[2] Univ Washington, Sch Med, Dept Med, Seattle, WA 98195 USA
[3] Univ Washington, Sch Med, Dept Neurol, Seattle, WA USA
基金
美国国家卫生研究院;
关键词
genetics; neurodegeneration; Parkinson disease; ALPHA-SYNUCLEIN GENE; EARLY-ONSET PARKINSONISM; AUTOSOMAL-DOMINANT PARKINSONISM; RECESSIVE JUVENILE PARKINSONISM; PROGRESSIVE SUPRANUCLEAR PALSY; PATHOGENIC DJ-1 MUTATIONS; GENOME-WIDE ASSOCIATION; MAPT H1 HAPLOTYPE; PINK1; MUTATIONS; LRRK2; G2019S;
D O I
10.1177/0891988710383572
中图分类号
R592 [老年病学]; C [社会科学总论];
学科分类号
03 ; 0303 ; 100203 ;
摘要
Parkinson disease (PD) is the second most common neurodegenerative disorder. In most instances, PD is thought to result from a complex interaction between multiple genetic and environmental factors, though rare monogenic forms of the disease do exist. Mutations in 6 genes (SNCA, LRRK2, PRKN, DJ1, PINK1, and ATP13A2) have conclusively been shown to cause familial parkinsonism. In addition, common variation in 3 genes (MAPT, LRRK2, and SNCA) and loss-of-function mutations in GBA have been well-validated as susceptibility factors for PD. The function of these genes and their contribution to PD pathogenesis remain to be fully elucidated. The prevalence, incidence, clinical manifestations, and genetic components of PD are discussed in this review.
引用
收藏
页码:228 / 242
页数:15
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