共 100 条
Sensing infection in Drosophila:: Toll and beyond
被引:86
作者:

Ferrandon, D
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机构:
CNRS, Inst Biol Mol & Cellulaire, UPR 9022, F-67084 Strasbourg, France CNRS, Inst Biol Mol & Cellulaire, UPR 9022, F-67084 Strasbourg, France

Imler, JL
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机构:
CNRS, Inst Biol Mol & Cellulaire, UPR 9022, F-67084 Strasbourg, France CNRS, Inst Biol Mol & Cellulaire, UPR 9022, F-67084 Strasbourg, France

Hoffmann, JA
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h-index: 0
机构:
CNRS, Inst Biol Mol & Cellulaire, UPR 9022, F-67084 Strasbourg, France CNRS, Inst Biol Mol & Cellulaire, UPR 9022, F-67084 Strasbourg, France
机构:
[1] CNRS, Inst Biol Mol & Cellulaire, UPR 9022, F-67084 Strasbourg, France
关键词:
Toll;
peptidoglycan recognition protein;
gram-negative binding protein;
beta-glucan recognition protein;
pattern recognition receptor;
D O I:
10.1016/j.smim.2003.10.008
中图分类号:
R392 [医学免疫学];
Q939.91 [免疫学];
学科分类号:
100102 ;
摘要:
Drosophila has evolved a potent immune system that is somewhat adapted to the nature of infections through the selective activation of either one of two NF-kappaB-like signalling pathways, the Toll and IMD (Immune deficiency) pathways. In contrast to the mammalian system, the Toll receptor does not act as a pattern recognition receptor (PRR) but as a cytokine receptor. The sensing of microbial infections is achieved by at least four PRRs that belong to two distinct families: the peptidoglycan recognition proteins (PGRPs) and the Gram-negative binding proteins (GNBPs)/beta-glucan recognition proteins (PGRPs). (C) 2003 Elsevier Ltd. All rights reserved.
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页码:43 / 53
页数:11
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