The extracellular matrix in normal and diseased myocardium

[31] EXPRESSION OF FIBRONECTIN DURING RAT FETAL AND POSTNATAL-DEVELOPMENT - AN IN-SITU HYBRIDIZATION AND IMMUNOHISTOCHEMICAL STUDY [J]. CARDIOVASCULAR RESEARCH, 1994, 28 (11) : 1653 - 1661

[32] Morphology and significance of the left ventricular collagen network in young patients with hypertrophic cardiomyopathy and sudden cardiac death [J]. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 2000, 35 (01) : 36 - 44

[33] RELATING EXTRACELLULAR POTENTIALS AND THEIR DERIVATIVES TO ANISOTROPIC PROPAGATION AT A MICROSCOPIC LEVEL IN HUMAN CARDIAC-MUSCLE - EVIDENCE FOR ELECTRICAL UNCOUPLING OF SIDE-TO-SIDE FIBER-CONNECTIONS WITH INCREASING AGE [J]. CIRCULATION RESEARCH, 1986, 58 (03) : 356 - 371

[34] Cellular and extracellular remodeling with the development and recovery from tachycardia-induced cardiomyopathy: Changes in fibrillar collagen, myocyte adhesion capacity and proteoglycans [J]. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 1996, 28 (08) : 1591 - 1608

[35] Myocardial matrix degradation and metalloproteinase activation in the failing heart: a potential therapeutic target [J]. CARDIOVASCULAR RESEARCH, 2000, 46 (02) : 225 - 238

[36] Time-dependent changes in matrix metalloproteinase activity and expression during the progression of congestive heart failure - Relation to ventricular and myocyte function [J]. CIRCULATION RESEARCH, 1998, 82 (04) : 482 - 495

[37] Alterations in the local myocardial motion pattern in patients suffering from pressure overload due to aortic stenosis [J]. CIRCULATION, 1999, 100 (04) : 361 - 368

[38] Sun Y, 1998, ANN MED, V30, P3

[39] Molecular mechanisms of myocardial remodeling [J]. PHYSIOLOGICAL REVIEWS, 1999, 79 (01) : 215 - 262

[40] STRUCTURE AND BIOLOGICAL-ACTIVITY OF BASEMENT-MEMBRANE PROTEINS [J]. EUROPEAN JOURNAL OF BIOCHEMISTRY, 1989, 180 (03): : 487 - 502

[31] EXPRESSION OF FIBRONECTIN DURING RAT FETAL AND POSTNATAL-DEVELOPMENT - AN IN-SITU HYBRIDIZATION AND IMMUNOHISTOCHEMICAL STUDY [J]. CARDIOVASCULAR RESEARCH, 1994, 28 (11) : 1653 - 1661

[32] Morphology and significance of the left ventricular collagen network in young patients with hypertrophic cardiomyopathy and sudden cardiac death [J]. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 2000, 35 (01) : 36 - 44

[33] RELATING EXTRACELLULAR POTENTIALS AND THEIR DERIVATIVES TO ANISOTROPIC PROPAGATION AT A MICROSCOPIC LEVEL IN HUMAN CARDIAC-MUSCLE - EVIDENCE FOR ELECTRICAL UNCOUPLING OF SIDE-TO-SIDE FIBER-CONNECTIONS WITH INCREASING AGE [J]. CIRCULATION RESEARCH, 1986, 58 (03) : 356 - 371

[34] Cellular and extracellular remodeling with the development and recovery from tachycardia-induced cardiomyopathy: Changes in fibrillar collagen, myocyte adhesion capacity and proteoglycans [J]. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 1996, 28 (08) : 1591 - 1608

[35] Myocardial matrix degradation and metalloproteinase activation in the failing heart: a potential therapeutic target [J]. CARDIOVASCULAR RESEARCH, 2000, 46 (02) : 225 - 238

[36] Time-dependent changes in matrix metalloproteinase activity and expression during the progression of congestive heart failure - Relation to ventricular and myocyte function [J]. CIRCULATION RESEARCH, 1998, 82 (04) : 482 - 495

[37] Alterations in the local myocardial motion pattern in patients suffering from pressure overload due to aortic stenosis [J]. CIRCULATION, 1999, 100 (04) : 361 - 368

[38] Sun Y, 1998, ANN MED, V30, P3

[39] Molecular mechanisms of myocardial remodeling [J]. PHYSIOLOGICAL REVIEWS, 1999, 79 (01) : 215 - 262

[40] STRUCTURE AND BIOLOGICAL-ACTIVITY OF BASEMENT-MEMBRANE PROTEINS [J]. EUROPEAN JOURNAL OF BIOCHEMISTRY, 1989, 180 (03): : 487 - 502