Church baptizes Joseph and Perry. Eccentric views absolved

[1] Lack of apolipoprotein E dramatically reduces amyloid beta-peptide deposition [J]. NATURE GENETICS, 1997, 17 (03) : 263 - 264

[2] DELAYED-ONSET OF ALZHEIMERS-DISEASE WITH NONSTEROIDAL ANTIINFLAMMATORY AND HISTAMINE-H2 BLOCKING-DRUGS [J]. NEUROBIOLOGY OF AGING, 1995, 16 (04) : 523 - 530

[3] FRAUTSCHY SA, 1998, NEUROBIOL AGING S4, V19, pS144

[4] FRAUTSCHY SA, 2000, 20 ANN SOC NEUROSCIE, V26

[5] Grimaldi LME, 2000, ANN NEUROL, V47, P361, DOI 10.1002/1531-8249(200003)47:3<361::AID-ANA12>3.0.CO

[6] 2-N

[7] Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes [J]. NATURE MEDICINE, 1998, 4 (01) : 97 - 100

[8] Apolipoprotein E isoform-dependent amyloid deposition and neuritic degeneration in a mouse model of Alzheimer's disease [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2000, 97 (06) : 2892 - 2897

[9] Diffusible, nonfibrillar ligands derived from Aβ1-42 are potent central nervous system neurotoxins [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1998, 95 (11) : 6448 - 6453

[10] Gene-expression profile of the ageing brain in mice [J]. NATURE GENETICS, 2000, 25 (03) : 294 - 297

[1] Lack of apolipoprotein E dramatically reduces amyloid beta-peptide deposition [J]. NATURE GENETICS, 1997, 17 (03) : 263 - 264

[2] DELAYED-ONSET OF ALZHEIMERS-DISEASE WITH NONSTEROIDAL ANTIINFLAMMATORY AND HISTAMINE-H2 BLOCKING-DRUGS [J]. NEUROBIOLOGY OF AGING, 1995, 16 (04) : 523 - 530

[3] FRAUTSCHY SA, 1998, NEUROBIOL AGING S4, V19, pS144

[4] FRAUTSCHY SA, 2000, 20 ANN SOC NEUROSCIE, V26

[5] Grimaldi LME, 2000, ANN NEUROL, V47, P361, DOI 10.1002/1531-8249(200003)47:3<361::AID-ANA12>3.0.CO

[6] 2-N

[7] Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes [J]. NATURE MEDICINE, 1998, 4 (01) : 97 - 100

[8] Apolipoprotein E isoform-dependent amyloid deposition and neuritic degeneration in a mouse model of Alzheimer's disease [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2000, 97 (06) : 2892 - 2897

[9] Diffusible, nonfibrillar ligands derived from Aβ1-42 are potent central nervous system neurotoxins [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1998, 95 (11) : 6448 - 6453

[10] Gene-expression profile of the ageing brain in mice [J]. NATURE GENETICS, 2000, 25 (03) : 294 - 297