A novel role for XIAP in copper homeostasis through regulation of MURR1

被引:177
作者
Burstein, E
Ganesh, L
Dick, RD
van de Sluis, B
Wilkinson, JC
Klomp, LWJ
Wijmenga, C
Brewer, GJ
Nabel, GJ
Duckett, CS
机构
[1] Univ Michigan, Sch Med, Dept Pathol, Ann Arbor, MI 48109 USA
[2] Univ Michigan, Sch Med, Dept Internal Med, Ann Arbor, MI USA
[3] Univ Michigan, Sch Med, Dept Human Genet, Ann Arbor, MI USA
[4] Univ Michigan, Sch Med, Dept Biomed Genet, Ann Arbor, MI USA
[5] NIAID, Vaccine Res Ctr, NIH, Bethesda, MD 20892 USA
关键词
copper; MURR1; ubiquitin; XIAP;
D O I
10.1038/sj.emboj.7600031
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
XIAP is a potent suppressor of apoptosis that directly inhibits specific members of the caspase family of cysteine proteases. Here we demonstrate a novel role for XIAP in the control of intracellular copper levels. XIAP was found to interact with MURR1, a factor recently implicated in copper homeostasis. XIAP binds to MURR1 in a manner that is distinct from that utilized by XIAP to bind caspases, and consistent with this, MURR1 did not affect the antiapoptotic properties of XIAP. However, cells and tissues derived from Xiap-deficient mice were found to contain reduced copper levels, while suppression of MURR1 resulted in increased intracellular copper in cultured cells. Consistent with these opposing effects, XIAP was observed to negatively regulate MURR1 protein levels by the formation of K48 polyubiquitin chains on MURR1 that promote its degradation. These findings represent the first described phenotypic alteration in Xiap-deficient mice and demonstrate that XIAP can function through MURR1 to regulate copper homeostasis.
引用
收藏
页码:244 / 254
页数:11
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