Complete polarization of single intestinal epithelial cells upon activation of LKB1 by STRAD

被引:471
作者
Baas, AF
Kuipers, J
van der Wel, NN
Batlle, E
Koerten, HK
Peters, PJ
Clevers, HC
机构
[1] Ctr Biomed Genet, Hubrecht Lab, NL-3584 CT Utrecht, Netherlands
[2] Netherlands Canc Inst, NL-1066 CX Amsterdam, Netherlands
[3] Leiden Univ, Med Ctr, Dept Mol Cell Biol, NL-2300 RA Leiden, Netherlands
关键词
D O I
10.1016/S0092-8674(04)00114-X
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 [生物化学与分子生物学]; 081704 [应用化学];
摘要
The LKB1 gene encodes a serine/threonine kinase that is mutated in the Peutz-Jeghers cancer syndrome. LKB1 is homologous to the Par-4 polarity genes in C. elegans and D. melanogaster. We have previously reported the identification and characterization of an LKB1-specific adaptor protein, STRAD, which activates LKB1 and translocates it from nucleus to cytoplasm. We have now constructed intestinal epithelial cell lines in which inducible STRAD activates LKB1. Upon LKB1 activation, single cells rapidly remodel their actin cytoskeleton to form an apical brush border. The junctional proteins ZO-1 and p120 redistribute in a dotted circle peripheral to the brush border, in the absence of cell-cell contacts. Apical and basolateral markers sort to their respective membrane domains. We conclude that LKB1 can induce complete polarity in intestinal epithelial cells. In contrast to current thinking on polarization of simple epithelia, these cells can fully polarize in the absence of junctional cell-cell contacts.
引用
收藏
页码:457 / 466
页数:10
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