Trypanosoma cruzi invades synaptotagmin VII-deficient cells by a PI-3 kinase independent pathway

被引：7

作者：

Chakrabarti, S ^{[1
]}

Andrade, LO ^{[1
]}

Andrews, NW ^{[1
]}

机构：

[1] Yale Univ, Sch Med, Boyer Ctr Mol Med, Sect Microbial Pathogenesis, New Haven, CT 06510 USA

来源：

MOLECULAR AND BIOCHEMICAL PARASITOLOGY | 2005年 / 141卷 / 01期

关键词：

trypomastigote; lysosome; invasion; calcium;

D O I：

10.1016/j.molbiopara.2005.01.016

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

[No abstract available]

引用

页码：125 / 128

页数：4

共 14 条

[11] Activation of host cell phosphatidylinositol 3-kinases by Trypanosoma cruzi infection [J].

Todorov, AG ;

Einicker-Lamas, M ;

de Castro, SL ;

Oliveira, MM ;

Guilherme, A .

JOURNAL OF BIOLOGICAL CHEMISTRY, 2000, 275 (41) :32182-32186

[12] Trypanosoma cruzi:: Phosphatidylinositol 3-kinase and protein kinase B activation is associated with parasite invasion [J].

Wilkowsky, SE ;

Barbieri, MA ;

Stahl, P ;

Isola, ELD .

EXPERIMENTAL CELL RESEARCH, 2001, 264 (02) :211-218

[13] Host cell actin polymerization is required for cellular retention of Trypanosoma cruzi and early association with endosomal/lysosomal compartments [J].

Woolsey, AM ;

Burleigh, BA .

CELLULAR MICROBIOLOGY, 2004, 6 (09) :829-838

[14] Novel PI 3-kinase-dependent mechanisms of trypanosome invasion and vacuole maturation [J].

Woolsey, AM ;

Sunwoo, L ;

Petersen, CA ;

Brachmann, SM ;

Cantley, LC ;

Burleigh, BA .

JOURNAL OF CELL SCIENCE, 2003, 116 (17) :3611-3622

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