Focus on the locus

被引：1

作者：

Burke, WJ

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来源：

ARCHIVES OF NEUROLOGY | 2003年 / 60卷 / 10期

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D O I：

10.1001/archneur.60.10.1493-a

中图分类号：

R74 [神经病学与精神病学];

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页码：1493 / 1493

页数：1

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共 12 条

[1] Norepinephrine transmitter metabolite induces apoptosis in differentiated rat pheochromocytoma cells [J].

Burke, WJ ;

Schmitt, CA ;

Miller, C ;

Li, SW .

BRAIN RESEARCH, 1997, 760 (1-2) :290-293

[2] Norepinephrine transmitter metabolite generates free radicals and activates mitochondrial permeability transition: a mechanism for DOPEGAL-induced apoptosis [J].

Burke, WJ ;

Kristal, BS ;

Yu, BP ;

Li, SW ;

Lin, TS .

BRAIN RESEARCH, 1998, 787 (02) :328-332

[3] Accumulation of 3,4-dihydroxyphenylglycolaldehyde, the neurotoxic monoamine oxidase A metabolite of norepinephrine, in locus ceruleus cell bodies in Alzheimer's disease: mechanism of neuron death [J].

Burke, WJ ;

Li, SW ;

Schmitt, CA ;

Xia, P ;

Chung, HD ;

Gillespie, KN .

BRAIN RESEARCH, 1999, 816 (02) :633-637

[4]

Burke WJ, 2000, NEUROTOXIC FACTORS IN PARKINSON'S DISEASE AND RELATED DISORDERS, P167

[5] Catecholamine monoamine oxidase a metabolite in adrenergic neurons is cytotoxic in vivo [J].

Burke, WJ ;

Li, SW ;

Zahm, DS ;

Macarthur, H ;

Kolo, LL ;

Westfall, TC ;

Anwar, M ;

Glickstein, SB ;

Ruggiero, DA .

BRAIN RESEARCH, 2001, 891 (1-2) :218-227

[6] Norepinephrine transmitter metabolite is a selective cell death messenger in differentiated rat pheochromocytoma cells [J].

Burke, WJ ;

Schmitt, CA ;

Gillespie, KN ;

Li, SW .

BRAIN RESEARCH, 1996, 722 (1-2) :232-235

[7] BETA-AMYLOID FIBRILS INDUCE TAU-PHOSPHORYLATION AND LOSS OF MICROTUBULE-BINDING [J].

BUSCIGLIO, J ;

LORENZO, A ;

YEH, J ;

YANKNER, BA .

NEURON, 1995, 14 (04) :879-888

[8] Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis [J].

Kayed, R ;

Head, E ;

Thompson, JL ;

McIntire, TM ;

Milton, SC ;

Cotman, CW ;

Glabe, CG .

SCIENCE, 2003, 300 (5618) :486-489

[9] Time to focus on the locus [J].

Rye, D ;

DeLong, MR .

ARCHIVES OF NEUROLOGY, 2003, 60 (03) :320-320

[10] Vesicle permeabilization by protofibrillar α-synuclein:: Implications for the pathogenesis and treatment of Parkinson's disease [J].

Volles, MJ ;

Lee, SJ ;

Rochet, JC ;

Shtilerman, MD ;

Ding, TT ;

Kessler, JC ;

Lansbury, PT .

BIOCHEMISTRY, 2001, 40 (26) :7812-7819