APP at a glance

[11] A transcriptively active complex of APP with Fe65 and histone acetyltransferase Tip60 [J]. SCIENCE, 2001, 293 (5527) : 115 - 120

[12] Dissection of amyloid-β precursor protein-dependent transcriptional transactivation [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2004, 279 (23) : 24601 - 24611

[13] Visualization of APP dimerization and APP-Notch2 heterodimerization in living cells using bimolecular fluorescence complementation [J]. JOURNAL OF NEUROCHEMISTRY, 2006, 97 (01) : 30 - 43

[14] APP-BP1 inhibits Aβ42 levels by interacting with Presenilin-1 [J]. MOLECULAR NEURODEGENERATION, 2007, 2 (1)

[15] APP-BP1 mediates APP-induced apoptosis and DNA synthesis and is increased in Alzheimer's disease brain [J]. JOURNAL OF CELL BIOLOGY, 2003, 163 (01) : 27 - 33

[16] Reelin depletion in the entorhinal cortex of human amyloid precursor protein transgenic mice and humans with Alzheimer's disease [J]. JOURNAL OF NEUROSCIENCE, 2007, 27 (11) : 2727 - 2733

[17] The amyloid precursor protein (APP)-cytoplasmic fragment generated by γ-secretase is rapidly degraded but distributes partially in a nuclear fraction of neurones in culture [J]. JOURNAL OF NEUROCHEMISTRY, 2001, 78 (05) : 1168 - 1178

[18] De Strooper B, 2000, J CELL SCI, V113, P1857

[19] A presenilin-1-dependent γ-secretase-like protease mediates release of Notch intracellular domain [J]. NATURE, 1999, 398 (6727) : 518 - 522

[20] Insulin-degrading enzyme rapidly removes the β-amyloid precursor protein intracellular domain (AICD) [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2002, 277 (16) : 13389 - 13393

[11] A transcriptively active complex of APP with Fe65 and histone acetyltransferase Tip60 [J]. SCIENCE, 2001, 293 (5527) : 115 - 120

[12] Dissection of amyloid-β precursor protein-dependent transcriptional transactivation [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2004, 279 (23) : 24601 - 24611

[13] Visualization of APP dimerization and APP-Notch2 heterodimerization in living cells using bimolecular fluorescence complementation [J]. JOURNAL OF NEUROCHEMISTRY, 2006, 97 (01) : 30 - 43

[14] APP-BP1 inhibits Aβ42 levels by interacting with Presenilin-1 [J]. MOLECULAR NEURODEGENERATION, 2007, 2 (1)

[15] APP-BP1 mediates APP-induced apoptosis and DNA synthesis and is increased in Alzheimer's disease brain [J]. JOURNAL OF CELL BIOLOGY, 2003, 163 (01) : 27 - 33

[16] Reelin depletion in the entorhinal cortex of human amyloid precursor protein transgenic mice and humans with Alzheimer's disease [J]. JOURNAL OF NEUROSCIENCE, 2007, 27 (11) : 2727 - 2733

[17] The amyloid precursor protein (APP)-cytoplasmic fragment generated by γ-secretase is rapidly degraded but distributes partially in a nuclear fraction of neurones in culture [J]. JOURNAL OF NEUROCHEMISTRY, 2001, 78 (05) : 1168 - 1178

[18] De Strooper B, 2000, J CELL SCI, V113, P1857

[19] A presenilin-1-dependent γ-secretase-like protease mediates release of Notch intracellular domain [J]. NATURE, 1999, 398 (6727) : 518 - 522

[20] Insulin-degrading enzyme rapidly removes the β-amyloid precursor protein intracellular domain (AICD) [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2002, 277 (16) : 13389 - 13393