Activation of synovial fibroblasts in rheumatoid arthritis: lack of expression of the tumour suppressor PTEN at sites of invasive growth and destruction

被引:146
作者
Pap, T [1 ]
Franz, JK [1 ]
Hummel, KM [1 ]
Jeisy, E [1 ]
Gay, R [1 ]
Gay, S [1 ]
机构
[1] Univ Zurich Hosp, Dept Rheumatol, WHO, Collaborat Ctr Mol Biol & Novel Therapeut Strateg, CH-8091 Zurich, Switzerland
关键词
rheumatoid arthritis; synovial membrane; fibroblasts; PTEN tumour suppressor; severe combined immunodeficiency (SCID) mouse model; cartilage destruction; in situ hybridization;
D O I
10.1186/ar69
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
In the present study, we searched for mutant PTEN transcripts in aggressive rheumatoid arthritis synovial fibroblasts (RA-SF) and studied the expression of PTEN in RA. By automated sequencing, no evidence for the presence of mutant PTEN transcripts was found. However, in situ hybridization on RA synovium revealed a distinct expression pattern of PTEN, with negligible staining in the lining layer but abundant expression in the sublining. Normal synovial tissue exhibited homogeneous staining for PTEN. In cultured RA-SF, only 40% expressed PTEN. Co-implantation of RA-SF and normal human cartilage into severe combined immunodeficiency (SCID) mice showed only limited expression of PTEN, with no staining in those cells aggressively invading the cartilage. Although PTEN is not genetically altered in RA, these findings suggest that a lack of PTEN expression may constitute a characteristic feature of activated RA-SF in the lining, and may thereby contribute to the invasive behaviour of RA-SF by maintaining their aggressive phenotype at sites of cartilage destruction.
引用
收藏
页码:59 / 64
页数:6
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