The origin of isocortical development

被引：7

作者：

Aboitiz, F ^{[1
]}

机构：

[1] Univ Chile, Fac Med, ICBM, Programa Morfol, Santiago 7, Chile

来源：

TRENDS IN NEUROSCIENCES | 2001年 / 24卷 / 04期

关键词：

D O I：

10.1016/S0166-2236(00)01771-9

中图分类号：

Q189 [神经科学];

学科分类号：

071006 ;

摘要：

引用

页码：202 / 203

页数：2

共 16 条

[1] Feature article evolution of isocortical organization. A tentative scenario including roles of reelin, p35/cdk5 and the subplate zone [J].

Aboitiz, F .

CEREBRAL CORTEX, 1999, 9 (07) :655-661

[2] The evolution of cortical development. An hypothesis based on the role of the Reelin signaling pathway [J].

Bar, I ;

de Rouvroit, CL ;

Goffinet, AM .

TRENDS IN NEUROSCIENCES, 2000, 23 (12) :633-638

[3]

CAVINESS VS, 1988, CEREBRAL CORTEX, V7

[4] Mice lacking p35, a neuronal specific activator of Cdk5, display cortical lamination defects, seizures, and adult lethality [J].

Chae, T ;

Kwon, YT ;

Bronson, R ;

Dikkes, P ;

Li, E ;

Tsai, LH .

NEURON, 1997, 18 (01) :29-42

[5] Reelin binds α3β1 integrin and inhibits neuronal migration [J].

Dulabon, L ;

Olson, EC ;

Taglienti, MG ;

Eisenhuth, S ;

McGrath, B ;

Walsh, CA ;

Kreidberg, JA ;

Anton, ES .

NEURON, 2000, 27 (01) :33-44

[6] Cajal-Retzius cells, Reelin, and the formation of layers [J].

Frotscher, M .

CURRENT OPINION IN NEUROBIOLOGY, 1998, 8 (05) :570-575

[7]

Gilmore EC, 1998, J NEUROSCI, V18, P6370

[8] Regulation of N-cadherin-mediated adhesion by the p35-Cdk5 kinase [J].

Kwon, YT ;

Gupta, A ;

Zhou, Y ;

Nikolic, M ;

Tsai, LH .

CURRENT BIOLOGY, 2000, 10 (07) :363-372

[9]

Kwon YT, 1998, J COMP NEUROL, V395, P510, DOI 10.1002/(SICI)1096-9861(19980615)395:4<510::AID-CNE7>3.0.CO

[10]

2-4

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