Dietary fatty acids linking postprandial metabolic response and chronic diseases

被引：21

作者：

Ortega, Almudena ^{[1
]}

Varela, Lourdes M. ^{[1
]}

Bermudez, Beatriz ^{[1
]}

Lopez, Sergio ^{[1
]}

Abia, Rocio ^{[1
]}

Muriana, Francisco J. G. ^{[1
]}

机构：

[1] Inst Grasa CSIC, Lab Cellular & Mol Nutr, Seville 41012, Spain

来源：

FOOD & FUNCTION | 2012年 / 3卷 / 01期

关键词：

TRIACYLGLYCEROL-RICH LIPOPROTEINS; INSULIN SENSITIVITY; OLIVE OIL; OLEIC-ACID; LIPEMIA; CHOLESTEROL; GLUCOSE; HEALTH; RATIO; MEN;

D O I：

10.1039/c1fo10085h

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

070307 [化学生物学]; 071010 [生物化学与分子生物学];

摘要：

Chronic diseases are by far one of the main causes of mortality in the world. One of the current global recommendations to counteract disability and premature death resulting from chronic diseases is to decrease the consumption of energy-dense high-fat diets, particularly those rich in saturated fatty acids (SFA). The most effective replacement for SFA in terms of risk factor outcomes for chronic disease are polyunsaturated fatty acids (PUFA) and monounsaturated fatty acids (MUFA). The biochemical basis for healthy benefits of such a dietary pattern has been widely evaluated under fasting conditions. However, the increasing amount of data available from multiple studies suggest that the postprandial state, i.e., "the period that comprises and follows a meal'', plays an important, yet underappreciated, role in the genesis of numerous pathological conditions. In this review, the potential of MUFA, PUFA, and SFA to postprandially affect selected metabolic abnormalities related to chronic diseases is discussed.

引用

页码：22 / 27

页数：6

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