PI3K enters beta-testing

被引：8

作者：

Shaywitz, Adam J. ^{[1
,2
]}

Courtney, Kevin D. ^{[1
,3
]}

Patnaik, Akash ^{[1
,4
]}

Cantley, Lewis C. ^{[1
,5
]}

机构：

[1] Beth Israel Deaconess Med Ctr, Div Signal Transduct, Boston, MA 02215 USA

[2] Beth Israel Deaconess Med Ctr, Div Endocrinol, Boston, MA 02215 USA

[3] Dana Farber Canc Inst, Dept Med Oncol, Boston, MA 02115 USA

[4] Beth Israel Deaconess Med Ctr, Div Hematol Oncol, Boston, MA 02215 USA

[5] Harvard Univ, Sch Med, Dept Syst Biol, Boston, MA 02115 USA

来源：

CELL METABOLISM | 2008年 / 8卷 / 03期

关键词：

D O I：

10.1016/j.cmet.2008.08.011

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

Phosphoinositide-3-OH kinases (PI3K) are critical regulators of cell metabolism, growth, and survival. In a recent publication in Nature, Jia et al. (2008) identify specific functions of the p110 beta isoform of PI3K in glucose metabolism, cellular proliferation, and tumorigenesis.

引用

页码：179 / 181

页数：3

共 12 条

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