FoxO3 controls dangerous proteolytic liaisons

被引：24

作者：

Attaix, Didier ^{[1
,2
]}

Bechet, Daniel ^{[1
,2
]}

机构：

[1] INRA, Proteolysis Grp, UMR1019, F-63122 Ceyrat, France

[2] Ctr Rech Nutr Humaine, F-63009 Clermont Ferrand, France

来源：

CELL METABOLISM | 2007年 / 6卷 / 06期

关键词：

D O I：

10.1016/j.cmet.2007.11.005

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

FoxO3 regulates the transcription of critical components of the ubiquitin-proteasome system in muscle wasting. Two reports (Mammucari et al., 2007; Zhao et al., 2007) now implicate FoxO3 in the transcription of autophagy-related genes and provide the first direct evidence for a coordinated role of autophagy in muscle atrophy.

引用

页码：425 / 427

页数：3

共 12 条

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