Neuronal models to study amyloid precursor protein expression and processing in vitro

被引:7
作者
Annaert, W [1 ]
De Strooper, B [1 ]
机构
[1] Katholieke Univ Leuven VIB, Ctr Human Genet, Neuronal Cell Biol & Gene Transfer Lab, B-3000 Louvain, Belgium
来源
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE | 2000年 / 1502卷 / 01期
关键词
amyloid precursor protein; presenilin; semliki forest virus; hipyocampal neuron; in vitro model;
D O I
10.1016/S0925-4439(00)00032-6
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
[No abstract available]
引用
收藏
页码:53 / 62
页数:10
相关论文
共 112 条
[81]   PDGF B-CHAIN IN NEURONS OF THE CENTRAL-NERVOUS-SYSTEM, POSTERIOR PITUITARY, AND IN A TRANSGENIC MODEL [J].
SASAHARA, M ;
FRIES, JWU ;
RAINES, EW ;
GOWN, AM ;
WESTRUM, LE ;
FROSCH, MP ;
BONTHRON, DT ;
ROSS, R ;
COLLINS, T .
CELL, 1991, 64 (01) :217-227
[82]   Secreted amyloid beta-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease [J].
Scheuner, D ;
Eckman, C ;
Jensen, M ;
Song, X ;
Citron, M ;
Suzuki, N ;
Bird, TD ;
Hardy, J ;
Hutton, M ;
Kukull, W ;
Larson, E ;
LevyLahad, E ;
Viitanen, M ;
Peskind, E ;
Poorkaj, P ;
Schellenberg, G ;
Tanzi, R ;
Wasco, W ;
Lannfelt, L ;
Selkoe, D ;
Younkin, S .
NATURE MEDICINE, 1996, 2 (08) :864-870
[83]   CHIMERIC PRION PROTEIN EXPRESSION IN CULTURED-CELLS AND TRANSGENIC MICE [J].
SCOTT, MR ;
KOHLER, R ;
FOSTER, D ;
PRUSINER, SB .
PROTEIN SCIENCE, 1992, 1 (08) :986-997
[84]   The cell biology of β-amyloid precursor protein and presenilin in Alzheimer's disease [J].
Selkoe, DJ .
TRENDS IN CELL BIOLOGY, 1998, 8 (11) :447-453
[85]   Amyloid beta-protein and the genetics of Alzheimer's disease [J].
Selkoe, DJ .
JOURNAL OF BIOLOGICAL CHEMISTRY, 1996, 271 (31) :18295-18298
[86]   Contact-dependent inhibition of cortical neurite growth mediated by notch signaling [J].
Sestan, N ;
Artavanis-Tsakonas, S ;
Rakic, P .
SCIENCE, 1999, 286 (5440) :741-746
[87]   CLONING OF A GENE BEARING MISSENSE MUTATIONS IN EARLY-ONSET FAMILIAL ALZHEIMERS-DISEASE [J].
SHERRINGTON, R ;
ROGAEV, EI ;
LIANG, Y ;
ROGAEVA, EA ;
LEVESQUE, G ;
IKEDA, M ;
CHI, H ;
LIN, C ;
LI, G ;
HOLMAN, K ;
TSUDA, T ;
MAR, L ;
FONCIN, JF ;
BRUNI, AC ;
MONTESI, MP ;
SORBI, S ;
RAINERO, I ;
PINESSI, L ;
NEE, L ;
CHUMAKOV, I ;
POLLEN, D ;
BROOKES, A ;
SANSEAU, P ;
POLINSKY, RJ ;
WASCO, W ;
DASILVA, HAR ;
HAINES, JL ;
PERICAKVANCE, MA ;
TANZI, RE ;
ROSES, AD ;
FRASER, PE ;
ROMMENS, JM ;
STGEORGEHYSLOP, PH .
NATURE, 1995, 375 (6534) :754-760
[88]   PRODUCTION OF THE ALZHEIMER AMYLOID-BETA PROTEIN BY NORMAL PROTEOLYTIC PROCESSING [J].
SHOJI, M ;
GOLDE, TE ;
GHISO, J ;
CHEUNG, TT ;
ESTUS, S ;
SHAFFER, LM ;
CAI, XD ;
MCKAY, DM ;
TINTNER, R ;
FRANGIONE, B ;
YOUNKIN, SG .
SCIENCE, 1992, 258 (5079) :126-129
[89]   Cholesterol depletion inhibits the generation of β-amyloid in hippocampal neurons [J].
Simons, M ;
Keller, P ;
De Strooper, B ;
Beyreuther, K ;
Dotti, CG ;
Simons, K .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1998, 95 (11) :6460-6464
[90]  
Simons M, 1996, J NEUROSCI, V16, P899