Insulin-like growth factor receptor 1 (IGFR-1) is significantly associated with longer survival in non-small-cell lung cancer patients treated with gefitinib

被引:85
作者
Cappuzzo, F.
Toschi, L.
Tallini, G.
Ceresoli, G. L.
Domenichini, I.
Bartolini, S.
Finocchiaro, G.
Magrini, E.
Metro, G.
Cancellieri, A.
Trisolini, R.
Crino, L.
Bunn, P. A., Jr.
Santoro, A.
Franklin, W. A.
Varella-Garcia, M.
Hirsch, F. R.
机构
[1] Univ Colorado, Ctr Canc, Dept Med Med Oncol & Pathol, Aurora, CO USA
[2] Bellaria Maggiore Hosp, Dept Med Oncol, Bologna, Italy
[3] Ist Clin Humanitas, Dept Med Oncol, Rozzano, Italy
[4] CINECA Interuniv Consortium, Bologna, Italy
关键词
IGFR-1; PTEN; EGFR; tyrosine kinase inhibitor; gefitinib; non-small-cell lung cancer;
D O I
10.1093/annonc/mdl077
中图分类号
R73 [肿瘤学];
学科分类号
100214 ;
摘要
Background: The aim of the study was to assess whether loss of PTEN and expression of insulin-like growth factor receptor 1 (IGFR-1) could be responsible for intrinsic resistance to the tyrosine kinase inhibitor (TKI) gefitinib. Patients and methods: One hundred and twenty-four gefitinib-treated patients with advanced non-small-cell lung cancer (NSCLC) were analyzed for PTEN and IGFR-1 expression by immunohistochemistry. Results: IGFR-1 was evaluated in 77 patients and resulted positive in 30 (39.0%). IGFR-1 expression was not significantly associated with clinical or biological characteristics. No difference in response to gefitinib treatment (16.7% versus 12.8%, P = 0.74) and time to progression (2.6 versus 3.06 months, P = 0.83) was observed between IGFR-1+ and IGFR-1-. Median survival was significantly longer in IGFR-1+ patients (17.8 versus 7.3 months, P = 0.013). PTEN expression was successfully evaluated in 93 cases. Loss of PTEN was detected in 19 tumors (20.4%) and was not associated with any clinical or biological characteristic. No difference in terms of response, time to progression and survival was observed between PTEN+ and PTEN- patients. In multivariable analysis IGFR-1 negative status was significantly associated with higher risk of death (hazard ratio 2.21, P = 0.012). Conclusions: IGFR-1 expression and loss of PTEN are not associated with intrinsic resistance to gefitinib. Clinical relevance of these two biomarkers as determinant for acquired resistance, and the prognostic role of IGFR-1 expression in patients not exposed to TKIs should be evaluated further.
引用
收藏
页码:1120 / 1127
页数:8
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