Mechanisms of Disease: Scleroderma.

[1] Genetics and genomic studies in scleroderma (systemic sclerosis) [J]. RHEUMATIC DISEASE CLINICS OF NORTH AMERICA, 2008, 34 (01) : 17 - +

[2] Dual inhibition of c-abl and PDGF receptor signaling by dasatinib and nilotinib for the treatment of dermal fibrosis [J]. FASEB JOURNAL, 2008, 22 (07) : 2214 - 2222

[3] Allanore Y, 2007, CLIN EXP RHEUMATOL, V25, P60

[4] Identification of fetal DNA and cells in skin lesions from women with systemic sclerosis [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1998, 338 (17) : 1186 - 1191

[5] Increased expression of integrin αvβ3 contributes to the establishment of autocrine TGF-β signaling in scleroderma fibroblasts [J]. JOURNAL OF IMMUNOLOGY, 2005, 175 (11) : 7708 - 7718

[6] Circulating endothelial progenitor cells in systemic sclerosis: association with disease severity [J]. ANNALS OF THE RHEUMATIC DISEASES, 2008, 67 (10) : 1455 - 1460

[7] Redox-mediated activation of latent transforming growth factor-beta 1 [J]. MOLECULAR ENDOCRINOLOGY, 1996, 10 (09) : 1077 - 1083

[8] Baroni SS, 2006, NEW ENGL J MED, V354, P2667

[9] The role of the fibrocyte, a bone marrow-derived mesenchymal progenitor, in reactive and reparative fibroses [J]. LABORATORY INVESTIGATION, 2007, 87 (09) : 858 - 870

[10] Reactive oxygen and nitrogen intermediates increase transforming growth factor-β1 release from human epithelial alveolar cells through two different mechanisms [J]. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, 1999, 21 (01) : 128 - 136

[1] Genetics and genomic studies in scleroderma (systemic sclerosis) [J]. RHEUMATIC DISEASE CLINICS OF NORTH AMERICA, 2008, 34 (01) : 17 - +

[2] Dual inhibition of c-abl and PDGF receptor signaling by dasatinib and nilotinib for the treatment of dermal fibrosis [J]. FASEB JOURNAL, 2008, 22 (07) : 2214 - 2222

[3] Allanore Y, 2007, CLIN EXP RHEUMATOL, V25, P60

[4] Identification of fetal DNA and cells in skin lesions from women with systemic sclerosis [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1998, 338 (17) : 1186 - 1191

[5] Increased expression of integrin αvβ3 contributes to the establishment of autocrine TGF-β signaling in scleroderma fibroblasts [J]. JOURNAL OF IMMUNOLOGY, 2005, 175 (11) : 7708 - 7718

[6] Circulating endothelial progenitor cells in systemic sclerosis: association with disease severity [J]. ANNALS OF THE RHEUMATIC DISEASES, 2008, 67 (10) : 1455 - 1460

[7] Redox-mediated activation of latent transforming growth factor-beta 1 [J]. MOLECULAR ENDOCRINOLOGY, 1996, 10 (09) : 1077 - 1083

[8] Baroni SS, 2006, NEW ENGL J MED, V354, P2667

[9] The role of the fibrocyte, a bone marrow-derived mesenchymal progenitor, in reactive and reparative fibroses [J]. LABORATORY INVESTIGATION, 2007, 87 (09) : 858 - 870

[10] Reactive oxygen and nitrogen intermediates increase transforming growth factor-β1 release from human epithelial alveolar cells through two different mechanisms [J]. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY, 1999, 21 (01) : 128 - 136