Toward antiapoptosis as a new treatment modality

[21] C-ELEGANS CELL-SURVIVAL GENE CED-9 ENCODES A FUNCTIONAL HOMOLOG OF THE MAMMALIAN PROTOONCOGENE BCL-2 [J]. CELL, 1994, 76 (04) : 665 - 676

[22] Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress [J]. CELL, 1999, 97 (02) : 189 - 198

[23] ITOH G, 1995, AM J PATHOL, V146, P1325

[24] Angiotensin II induces apoptosis of adult ventricular myocytes in vitro [J]. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 1997, 29 (03) : 859 - 870

[25] Kajstura J, 1996, LAB INVEST, V74, P86

[26] Significance of myocytes with positive DNA in situ nick end-labeling (TUNEL) in hearts with dilated cardiomyopathy - Not apoptosis but DNA repair [J]. CIRCULATION, 1999, 99 (21) : 2757 - 2764

[27] BAX-DEFICIENT MICE WITH LYMPHOID HYPERPLASIA AND MALE GERM-CELL DEATH [J]. SCIENCE, 1995, 270 (5233) : 96 - 99

[28] ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1998, 95 (09) : 5156 - 5160

[29] Tumor necrosis factor alpha-induced apoptosis in cardiac myocytes - Involvement of the sphingolipid signaling cascade in cardiac cell death [J]. JOURNAL OF CLINICAL INVESTIGATION, 1996, 98 (12) : 2854 - 2865

[30] Dilated cardiomyopathy in transgenic mice with cardiac-specific overexpression of tumor necrosis factor-alpha [J]. CIRCULATION RESEARCH, 1997, 81 (04) : 627 - 635

[21] C-ELEGANS CELL-SURVIVAL GENE CED-9 ENCODES A FUNCTIONAL HOMOLOG OF THE MAMMALIAN PROTOONCOGENE BCL-2 [J]. CELL, 1994, 76 (04) : 665 - 676

[22] Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress [J]. CELL, 1999, 97 (02) : 189 - 198

[23] ITOH G, 1995, AM J PATHOL, V146, P1325

[24] Angiotensin II induces apoptosis of adult ventricular myocytes in vitro [J]. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY, 1997, 29 (03) : 859 - 870

[25] Kajstura J, 1996, LAB INVEST, V74, P86

[26] Significance of myocytes with positive DNA in situ nick end-labeling (TUNEL) in hearts with dilated cardiomyopathy - Not apoptosis but DNA repair [J]. CIRCULATION, 1999, 99 (21) : 2757 - 2764

[27] BAX-DEFICIENT MICE WITH LYMPHOID HYPERPLASIA AND MALE GERM-CELL DEATH [J]. SCIENCE, 1995, 270 (5233) : 96 - 99

[28] ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1998, 95 (09) : 5156 - 5160

[29] Tumor necrosis factor alpha-induced apoptosis in cardiac myocytes - Involvement of the sphingolipid signaling cascade in cardiac cell death [J]. JOURNAL OF CLINICAL INVESTIGATION, 1996, 98 (12) : 2854 - 2865

[30] Dilated cardiomyopathy in transgenic mice with cardiac-specific overexpression of tumor necrosis factor-alpha [J]. CIRCULATION RESEARCH, 1997, 81 (04) : 627 - 635