Responses by murine macrophages infected with Listeria monocytogenes crucial for the development of immunity to this pathogen

被引：13

作者：

Kuhn, M

Goebel, W

机构：

[1] Lehrstuhl für Mikrobiologie, Theodor-Boveri-Inst. F. B., Universität Würzburg, Würzburg

[2] Lehrstuhl für Mikrobiologie, Theodor-Boveri-Inst. F. B., Universität Würzburg, 97074 Würzburg, Am Hubland

来源：

IMMUNOLOGICAL REVIEWS | 1997年 / 158卷

关键词：

D O I：

10.1111/j.1600-065X.1997.tb00992.x

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

Macrophages and other mammalian cells respond to infection with Listeria monocytogenes (L. monocytogenes) by the transient or persistent activation of host cell signal transduction pathways. In addition, L. monocytogenes infection influences expression of various host cell genes, such as stress genes, genes from the MHC I and II complex, cytokine genes, and cytokine receptor genes. The possible influences of the different host cell responses on the outcome of an L. monocytogenes infection in vitro as well as for the development of immunity are discussed.

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页码：57 / 67

页数：11

相关论文

共 87 条

[11] INTRACELLULAR HEMOLYSIN-PRODUCING LISTERIA-MONOCYTOGENES STRAINS INHIBIT MACROPHAGE-MEDIATED ANTIGEN PROCESSING [J].

CLUFF, CW ;

GARCIA, M ;

ZIEGLER, HK .

INFECTION AND IMMUNITY, 1990, 58 (11) :3601-3612

[12] ACTIVATION OF THE HUMAN-COMPLEMENT ALTERNATIVE PATHWAY BY LISTERIA-MONOCYTOGENES - EVIDENCE FOR DIRECT BINDING AND PROTEOLYSIS OF THE C3 COMPONENT ON BACTERIA [J].

CROIZE, J ;

ARVIEUX, J ;

BERCHE, P ;

COLOMB, MG .

INFECTION AND IMMUNITY, 1993, 61 (12) :5134-5139

[13] Differential regulation of cytokine and cytokine receptor mRNA expression upon infection of bone marrow-derived macrophages with Listeria monocytogenes [J].

Demuth, A ;

Goebel, W ;

Beuscher, HU ;

Kuhn, M .

INFECTION AND IMMUNITY, 1996, 64 (09) :3475-3483

[14] MAMMALIAN-CELLS TRANSFECTED WITH THE LISTERIOLYSIN GENE EXHIBIT ENHANCED PROLIFERATION AND FOCUS FORMATION [J].

DEMUTH, A ;

CHAKRABORTY, T ;

KROHNE, G ;

GOEBEL, W .

INFECTION AND IMMUNITY, 1994, 62 (11) :5102-5111

[15] Biologic basis for interleukin-1 in disease [J].

Dinarello, CA .

BLOOD, 1996, 87 (06) :2095-2147

[16] A NOVEL BACTERIAL VIRULENCE GENE IN LISTERIA-MONOCYTOGENES REQUIRED FOR HOST-CELL MICROFILAMENT INTERACTION WITH HOMOLOGY TO THE PROLINE-RICH REGION OF VINCULIN [J].

DOMANN, E ;

WEHLAND, J ;

ROHDE, M ;

PISTOR, S ;

HARTL, M ;

GOEBEL, W ;

LEIMEISTERWACHTER, M ;

WUENSCHER, M ;

CHAKRABORTY, T .

EMBO JOURNAL, 1992, 11 (05) :1981-1990

[17]

DRAMSI S, 1995, MOL MICROBIOL, V16, P251, DOI 10.1111/j.1365-2958.1995.tb02297.x

[18] Complement receptor type 3 mediates phagocytosis and killing of Listeria monocytogenes by a TNF-alpha- and IFN-gamma-stimulated macrophage precursor hybrid [J].

Drevets, DA ;

Leenen, PJM ;

Campbell, PA .

CELLULAR IMMUNOLOGY, 1996, 169 (01) :1-6

[19] LISTERICIDAL AND NONLISTERICIDAL MOUSE MACROPHAGES DIFFER IN COMPLEMENT RECEPTOR TYPE 3-MEDIATED PHAGOCYTOSIS OF L-MONOCYTOGENES AND IN PREVENTING ESCAPE OF THE BACTERIA INTO THE CYTOPLASM [J].

DREVETS, DA ;

CANONO, BP ;

CAMPBELL, PA .

JOURNAL OF LEUKOCYTE BIOLOGY, 1992, 52 (01) :70-79

[20]

DREVETS DA, 1993, J IMMUNOL, V151, P5431

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