Resveratrol and cancer: a review

被引:169
作者
Savouret, JF [1 ]
Quesne, M [1 ]
机构
[1] CHU Le Kremlin Bicetre, Hop Bicetre, INSERM U135, F-94270 Le Kremlin Bicetre, France
关键词
aryl hydrocarbon receptor; chemoprevention; resveratrol;
D O I
10.1016/S0753-3322(01)00158-5
中图分类号
R-3 [医学研究方法]; R3 [基础医学];
学科分类号
1001 ;
摘要
The various properties of the stilbene phytoalexin Resveratrol provide interesting new avenues of research in the field of chemoprevention and chemotherapy. A particular emphasis is given on xenobiotic-related carcinogenesis. (C) 2002 Editions scientifiques et medicales Elsevier SAS.
引用
收藏
页码:84 / 87
页数:4
相关论文
共 40 条
[21]   Chemoprevention by inducers of carcinogen detoxication enzymes [J].
Kensler, TW .
ENVIRONMENTAL HEALTH PERSPECTIVES, 1997, 105 :965-970
[22]   PRODUCTION OF RESVERATROL BY VITIS-VINIFERA AND OTHER MEMBERS OF VITACEAE AS A RESPONSE TO INFECTION OR INJURY [J].
LANGCAKE, P ;
PRYCE, RJ .
PHYSIOLOGICAL PLANT PATHOLOGY, 1976, 9 (01) :77-86
[23]   4-HYDROXYLATION OF ESTRADIOL BY HUMAN UTERINE MYOMETRIUM AND MYOMA MICROSOMES - IMPLICATIONS FOR THE MECHANISM OF UTERINE TUMORIGENESIS [J].
LIEHR, JG ;
RICCI, MJ ;
JEFCOATE, CR ;
HANNIGAN, EV ;
HOKANSON, JA ;
ZHU, BT .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (20) :9220-9224
[24]   Resveratrol analog, 3,4,5,4′-tetrahydroxystilbene, differentially induces pro-apoptotic p53/Bax gene expression and inhibits the growth of transformed cells but not their normal counterparts [J].
Lu, JB ;
Ho, CT ;
Ghai, G ;
Chen, KY .
CARCINOGENESIS, 2001, 22 (02) :321-328
[25]  
Lu RQ, 1999, J CELL PHYSIOL, V179, P297, DOI 10.1002/(SICI)1097-4652(199906)179:3<297::AID-JCP7>3.0.CO
[26]  
2-P
[27]   Resveratrol prevents apoptosis in K562 cells by inhibiting lipoxygenase and cyclooxygenase activity [J].
Maccarrone, M ;
Lorenzon, T ;
Guerrieri, P ;
Agró, AF .
EUROPEAN JOURNAL OF BIOCHEMISTRY, 1999, 265 (01) :27-34
[28]  
Maggiolini M, 2001, MOL PHARMACOL, V60, P595
[29]  
NONOMURA S, 1963, YAKUGAKU ZASSHI, V83, P983
[30]   Chronic immune activation and inflammation as the cause of malignancy [J].
O'Byrne, KJ ;
Dalgleish, A .
BRITISH JOURNAL OF CANCER, 2001, 85 (04) :473-483