Tubulointerstitium as predictor of progression of glomerular diseases

被引：59

作者：

D'Amico, G ^{[1
]}

机构：

[1] San Carlo Borromeo Hosp, Div Nephrol, I-20153 Milan, Italy

来源：

NEPHRON | 1999年 / 83卷 / 04期

关键词：

D O I：

10.1159/000045419

中图分类号：

R5 [内科学]; R69 [泌尿科学（泌尿生殖系疾病）];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：289 / 295

页数：7

共 82 条

[71] ON THE PROGRESSION OF CHRONIC RENAL-DISEASE [J].

STRUTZ, F ;

MULLER, GA .

NEPHRON, 1995, 69 (04) :371-379

[72]

STRUTZ F, 1995, NEPHROL DIAL TRANSPL, V10, P1526

[73]

STRUTZ F, 1994, J AM SOC NEPHROL, V5, P819

[74]

Strutz F, 1994, NEPHROL DIAL TRANS S, V9, P10

[75]

Tang W. W., 1996, Journal of the American Society of Nephrology, V7, P1766

[76] PRODUCTION OF COLLAGEN TYPE-I BY MOUSE PERITONEAL-MACROPHAGES [J].

VAAGE, J ;

LINDBLAD, WJ .

JOURNAL OF LEUKOCYTE BIOLOGY, 1990, 48 (03) :274-280

[77] Possible role for CD40-CD40L in the regulation of interstitial infiltration in the kidney [J].

vanKooten, C ;

Gerritsma, JSJ ;

Paape, ME ;

vanEs, LA ;

Banchereau, J ;

Daha, MR .

KIDNEY INTERNATIONAL, 1997, 51 (03) :711-721

[78] LOCALIZATION OF PROLIFERATING CELL NUCLEAR ANTIGEN, VIMENTIN, C-FOS, AND CLUSTERIN IN THE POSTISCHEMIC KIDNEY - EVIDENCE FOR A HETEROGENOUS GENETIC RESPONSE AMONG NEPHRON SEGMENTS, AND A LARGE POOL OF MITOTICALLY ACTIVE AND DEDIFFERENTIATED CELLS [J].

WITZGALL, R ;

BROWN, D ;

SCHWARZ, C ;

BONVENTRE, JV .

JOURNAL OF CLINICAL INVESTIGATION, 1994, 93 (05) :2175-2188

[79] ANGIOTENSIN-II-INDUCED HYPERTROPHY OF CULTURED MURINE PROXIMAL TUBULAR CELLS IS MEDIATED BY ENDOGENOUS TRANSFORMING GROWTH-FACTOR-BETA [J].

WOLF, G ;

MUELLER, E ;

STAHL, RAK ;

ZIYADEH, FN .

JOURNAL OF CLINICAL INVESTIGATION, 1993, 92 (03) :1366-1372

[80] MHC CLASS-II, ANTIGEN PRESENTATION AND TUMOR NECROSIS FACTOR IN RENAL TUBULAR EPITHELIAL-CELLS [J].

WUTHRICH, RP ;

GLIMCHER, LH ;

YUI, MA ;

JEVNIKAR, AM ;

DUMAS, SE ;

KELLEY, VE .

KIDNEY INTERNATIONAL, 1990, 37 (02) :783-792

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