Serpinopathies and the conformational dementias

被引:187
作者
Lomas, DA
Carrell, RW
机构
[1] Univ Cambridge, Cambridge Inst Med Res, Dept Med, Cambridge CB2 2XY, England
[2] Univ Cambridge, Cambridge Inst Med Res, Dept Haematol, Cambridge CB2 2XY, England
基金
英国医学研究理事会; 英国惠康基金;
关键词
D O I
10.1038/nrg907
中图分类号
Q3 [遗传学];
学科分类号
071007 ; 090102 ;
摘要
The serpin superfamily of serine proteinase inhibitors has a central role in controlling proteinases in many biological pathways in a wide range of species. The inhibitory function of the serpins involves a marked conformational transition, but this inherent molecular flexibility also renders the serpins susceptible to point mutations that result in aberrant intermolecular linkage and polymer formation. The effects of such protein aggregation are cumulative, with a progressive loss of cellular function that results in diseases as diverse as cirrhosis and emphysema. The recent recognition that mutations in a serpin can also result in late-onset dementia provides insights into changes that underlie other conformational diseases, such as the amyloidoses, the prion encephalopathies and Huntington and Alzheimer diseases.
引用
收藏
页码:759 / 768
页数:10
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