Vascular endothelial growth factor-C accelerates diabetic wound healing

被引:197
作者
Saaristo, Anne
Tammela, Tuomas
Farkkila, Anniina
Karkkainen, Marika
Suominen, Erkki
Yla-Herttuala, Seppo
Alitalo, Kari
机构
[1] Univ Helsinki, Biomedicum Helsinki, Lab Mol Canc Biol, FIN-00014 Helsinki, Finland
[2] Univ Helsinki, Biomedicum Helsinki, Ludwig Inst Canc Res, FIN-00014 Helsinki, Finland
[3] Univ Helsinki, Cent Hosp, FIN-00014 Helsinki, Finland
[4] Turku Univ, Cent Hosp, Dept Plast Surg, Turku, Finland
[5] Univ Kuopio, AI Virtanen Inst, Dept Med, FIN-70211 Kuopio, Finland
[6] Univ Kuopio, AI Virtanen Inst, Gene Therapy Unit, FIN-70211 Kuopio, Finland
关键词
D O I
10.2353/ajpath.2006.051251
中图分类号
R36 [病理学];
学科分类号
100104 ;
摘要
Diabetes impairs numerous aspects of tissue repair. Failure of wound angiogenesis is known to delay diabetic wound healing, whereas the importance of lymphangiogenesis for wound healing is unclear. We have examined whether overexpression of vascular endothelial growth factor (VEGF)-C via an adenoviral vector could improve the healing of full-thickness punch biopsy wounds in genetically diabetic (db/db) mice. We found that VEGF-C enhanced angiogenesis and lymphangiogenesis in the wound and significantly accelerated wound healing in comparison to the control wounds. VEGF-C also recruited inflammatory cells, some of which expressed VEGFR-3. On the other hand, when the function of endogenous VEGF-C/VEGF-D was blocked with a specific inhibitor, wound closure was delayed even further. These results suggest a function for VEGF-C in wound healing and demonstrate the therapeutic potential of VEGF-C in the treatment of diabetic wounds.
引用
收藏
页码:1080 / 1087
页数:8
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