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Protein degradation by the ubiquitin-proteasome pathway in normal and disease states
被引:925
作者
:
Lecker, Stewart H.
论文数:
0
引用数:
0
h-index:
0
机构:
Baylor Coll Med, Nephrol Div MS, Houston, TX 77030 USA
Lecker, Stewart H.
Goldberg, Alfred L.
论文数:
0
引用数:
0
h-index:
0
机构:
Baylor Coll Med, Nephrol Div MS, Houston, TX 77030 USA
Goldberg, Alfred L.
Mitch, William E.
论文数:
0
引用数:
0
h-index:
0
机构:
Baylor Coll Med, Nephrol Div MS, Houston, TX 77030 USA
Mitch, William E.
机构
:
[1]
Baylor Coll Med, Nephrol Div MS, Houston, TX 77030 USA
[2]
Harvard Univ, Sch Med, Div Nephrol, Boston, MA 02115 USA
[3]
Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA
来源
:
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
|
2006年
/ 17卷
/ 07期
关键词
:
D O I
:
10.1681/ASN.2006010083
中图分类号
:
R5 [内科学];
R69 [泌尿科学(泌尿生殖系疾病)];
学科分类号
:
1002 ;
100201 ;
摘要
:
[No abstract available]
引用
收藏
页码:1807 / 1819
页数:13
相关论文
共 110 条
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The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway
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Bailey, JL
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共 110 条
[1]
The proteasome: A suitable antineoplastic target
[J].
Adams, J
论文数:
0
引用数:
0
h-index:
0
机构:
Infin Pharmaceut Inc, Cambridge, MA 02139 USA
Infin Pharmaceut Inc, Cambridge, MA 02139 USA
Adams, J
.
NATURE REVIEWS CANCER,
2004,
4
(05)
:349
-360
[2]
Imprinted expression of the murine Angelman syndrome gene, Ube3a, in hippocampal and Purkinje neurons
[J].
Albrecht, U
论文数:
0
引用数:
0
h-index:
0
机构:
BAYLOR COLL MED, DEPT MOL & HUMAN GENET, HOUSTON, TX 77030 USA
Albrecht, U
;
Sutcliffe, JS
论文数:
0
引用数:
0
h-index:
0
机构:
BAYLOR COLL MED, DEPT MOL & HUMAN GENET, HOUSTON, TX 77030 USA
Sutcliffe, JS
;
Cattanach, BM
论文数:
0
引用数:
0
h-index:
0
机构:
BAYLOR COLL MED, DEPT MOL & HUMAN GENET, HOUSTON, TX 77030 USA
Cattanach, BM
;
Beechey, CV
论文数:
0
引用数:
0
h-index:
0
机构:
BAYLOR COLL MED, DEPT MOL & HUMAN GENET, HOUSTON, TX 77030 USA
Beechey, CV
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Armstrong, D
论文数:
0
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0
h-index:
0
机构:
BAYLOR COLL MED, DEPT MOL & HUMAN GENET, HOUSTON, TX 77030 USA
Armstrong, D
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论文数:
0
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0
h-index:
0
机构:
BAYLOR COLL MED, DEPT MOL & HUMAN GENET, HOUSTON, TX 77030 USA
Eichele, G
;
Beaudet, AL
论文数:
0
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机构:
BAYLOR COLL MED, DEPT MOL & HUMAN GENET, HOUSTON, TX 77030 USA
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h-index:
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Univ Maryland, Maryland Biotechnol Inst, Ctr Biosyst Res, College Pk, MD 20742 USA
Baehrecke, EH
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NATURE REVIEWS MOLECULAR CELL BIOLOGY,
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The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway
[J].
Bailey, JL
论文数:
0
引用数:
0
h-index:
0
机构:
Department of Medicine, Renal Division, Emory University School of Medicine, Atlanta
Bailey, JL
;
Wang, XN
论文数:
0
引用数:
0
h-index:
0
机构:
Department of Medicine, Renal Division, Emory University School of Medicine, Atlanta
Wang, XN
;
England, BK
论文数:
0
引用数:
0
h-index:
0
机构:
Department of Medicine, Renal Division, Emory University School of Medicine, Atlanta
England, BK
;
Price, SR
论文数:
0
引用数:
0
h-index:
0
机构:
Department of Medicine, Renal Division, Emory University School of Medicine, Atlanta
Price, SR
;
Ding, XY
论文数:
0
引用数:
0
h-index:
0
机构:
Department of Medicine, Renal Division, Emory University School of Medicine, Atlanta
Ding, XY
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Mitch, WE
论文数:
0
引用数:
0
h-index:
0
机构:
Department of Medicine, Renal Division, Emory University School of Medicine, Atlanta
Mitch, WE
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Baumeister, W
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0
引用数:
0
h-index:
0
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Max Planck Inst Biochem, D-82152 Martinsried, Germany
Max Planck Inst Biochem, D-82152 Martinsried, Germany
Baumeister, W
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Walz, J
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0
引用数:
0
h-index:
0
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Max Planck Inst Biochem, D-82152 Martinsried, Germany
Max Planck Inst Biochem, D-82152 Martinsried, Germany
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0
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0
h-index:
0
机构:
Max Planck Inst Biochem, D-82152 Martinsried, Germany
Max Planck Inst Biochem, D-82152 Martinsried, Germany
Zühl, F
;
Seemuller, E
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0
引用数:
0
h-index:
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Max Planck Inst Biochem, D-82152 Martinsried, Germany
Max Planck Inst Biochem, D-82152 Martinsried, Germany
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Benaroudj, N
论文数:
0
引用数:
0
h-index:
0
机构:
Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA
Benaroudj, N
;
Zwickl, P
论文数:
0
引用数:
0
h-index:
0
机构:
Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA
Zwickl, P
;
Seemüller, E
论文数:
0
引用数:
0
h-index:
0
机构:
Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA
Seemüller, E
;
Baumeister, W
论文数:
0
引用数:
0
h-index:
0
机构:
Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA
Baumeister, W
;
Goldberg, AL
论文数:
0
引用数:
0
h-index:
0
机构:
Harvard Univ, Sch Med, Dept Cell Biol, Boston, MA 02115 USA
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MOLECULAR CELL,
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[8]
Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo
[J].
Bodine, SC
论文数:
0
引用数:
0
h-index:
0
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Regeneron Pharmaceut Inc, Tarrytown, NY 10591 USA
Bodine, SC
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Stitt, TN
论文数:
0
引用数:
0
h-index:
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Regeneron Pharmaceut Inc, Tarrytown, NY 10591 USA
Stitt, TN
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Gonzalez, M
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0
引用数:
0
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0
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Regeneron Pharmaceut Inc, Tarrytown, NY 10591 USA
Gonzalez, M
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Kline, WO
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0
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h-index:
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Regeneron Pharmaceut Inc, Tarrytown, NY 10591 USA
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Regeneron Pharmaceut Inc, Tarrytown, NY 10591 USA
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机构:
Regeneron Pharmaceut Inc, Tarrytown, NY 10591 USA
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Regeneron Pharmaceut Inc, Tarrytown, NY 10591 USA
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Regeneron Pharmaceut Inc, Tarrytown, NY 10591 USA
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