BRCA1, BRCA2, and DNA damage response: Collision or collusion?

[1] Transcriptional activation by BRCA1 [J]. NATURE, 1996, 382 (6593) : 678 - 679

[2] Tumorigenesis and a DNA repair defect in mice with a truncating Brca2 mutation [J]. NATURE GENETICS, 1997, 17 (04) : 423 - 430

[3] p53 mutations in BRCA1-associated familial breast cancer [J]. LANCET, 1997, 350 (9078) : 638 - 639

[4] THE BLOOMS-SYNDROME GENE-PRODUCT IS HOMOLOGOUS TO RECQ HELICASES [J]. CELL, 1995, 83 (04) : 655 - 666

[5] FREIDBERG EC, 1995, DNA REPAIR MUTAGENES

[6] FRIEDMAN LS, 1998, IN PRESS CANC RES

[7] Lim DS, 1996, MOL CELL BIOL, V16, P7133

[8] Targeted mutations of breast cancer susceptibility gene homologs in mice: lethal phenotypes of Brca1, Brca2, Brca1/Brca2, Brca1/p53, and Brca2/p53 nullizygous embryos [J]. GENES & DEVELOPMENT, 1997, 11 (10) : 1226 - 1241

[9] A human RNA polymerase II complex associated with SRB and DNA-repair proteins [J]. NATURE, 1996, 381 (6577) : 86 - 89

[10] hMre11 and hRad50 nuclear foci are induced during the normal cellular response to DNA double-strand breaks [J]. MOLECULAR AND CELLULAR BIOLOGY, 1997, 17 (10) : 6087 - 6096

[1] Transcriptional activation by BRCA1 [J]. NATURE, 1996, 382 (6593) : 678 - 679

[2] Tumorigenesis and a DNA repair defect in mice with a truncating Brca2 mutation [J]. NATURE GENETICS, 1997, 17 (04) : 423 - 430

[3] p53 mutations in BRCA1-associated familial breast cancer [J]. LANCET, 1997, 350 (9078) : 638 - 639

[4] THE BLOOMS-SYNDROME GENE-PRODUCT IS HOMOLOGOUS TO RECQ HELICASES [J]. CELL, 1995, 83 (04) : 655 - 666

[5] FREIDBERG EC, 1995, DNA REPAIR MUTAGENES

[6] FRIEDMAN LS, 1998, IN PRESS CANC RES

[7] Lim DS, 1996, MOL CELL BIOL, V16, P7133

[8] Targeted mutations of breast cancer susceptibility gene homologs in mice: lethal phenotypes of Brca1, Brca2, Brca1/Brca2, Brca1/p53, and Brca2/p53 nullizygous embryos [J]. GENES & DEVELOPMENT, 1997, 11 (10) : 1226 - 1241

[9] A human RNA polymerase II complex associated with SRB and DNA-repair proteins [J]. NATURE, 1996, 381 (6577) : 86 - 89

[10] hMre11 and hRad50 nuclear foci are induced during the normal cellular response to DNA double-strand breaks [J]. MOLECULAR AND CELLULAR BIOLOGY, 1997, 17 (10) : 6087 - 6096