A positive feedback loop bi-stably activates fibroblasts

被引:89
作者
Yeo, So-Young [1 ]
Lee, Keun-Woo [1 ]
Shin, Dongkwan [2 ]
An, Sugyun [2 ]
Cho, Kwang-Hyun [2 ]
Kim, Seok-Hyung [1 ,3 ,4 ]
机构
[1] Sungkyunkwan Univ, Samsung Adv Inst Hlth Sci & Technol, Dept Hlth Sci & Technol, Seoul 06351, South Korea
[2] Korea Adv Inst Sci & Technol, Dept Bio & Brain Engn, Daejeon 34141, South Korea
[3] Sungkyunkwan Univ, Sch Med, Samsung Med Ctr, Dept Pathol, Seoul 06351, South Korea
[4] Sungkyunkwan Univ, Sch Med, Single Cell Network Res Ctr, Suwona 16419, Gyeonggi Do, South Korea
基金
新加坡国家研究基金会;
关键词
CANCER-ASSOCIATED FIBROBLASTS; TENASCIN-C; TRANSCRIPTION FACTOR; MONOCLONAL-ANTIBODY; SIGNAL-TRANSDUCTION; PULMONARY-FIBROSIS; PHASE-II; MATRIX; TWIST1; TISSUE;
D O I
10.1038/s41467-018-05274-6
中图分类号
O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];
学科分类号
070301 [无机化学]; 070403 [天体物理学]; 070507 [自然资源与国土空间规划学]; 090105 [作物生产系统与生态工程];
摘要
Although fibroblasts are dormant in normal tissue, they exhibit explosive activation during wound healing and perpetual activation in pathologic fibrosis and cancer stroma. The key regulatory network controlling these fibroblast dynamics is still unknown. Here, we report that Twist1, a key regulator of cancer-associated fibroblasts, directly upregulates Prrx1, which, in turn, increases the expression of Tenascin-C (TNC). TNC also increases Twist1 expression, consequently forming a Twist1-Prrx1-TNC positive feedback loop (PFL). Systems biology studies reveal that the Twist1-Prrx1-TNC PFL can function as a bistable ON/OFF switch and regulates fibroblast activation. This PFL can be irreversibly activated under pathologic conditions, leading to perpetual fibroblast activation. Sustained activation of the Twist1-Prrx1-TNC PFL reproduces fibrotic nodules similar to idiopathic pulmonary fibrosis in vivo and is implicated in fibrotic disease and cancer stroma. Considering that this PFL is specific to activated fibroblasts, Twist1-Prrx1-TNC PFL may be a fibroblast-specific therapeutic target to deprogram perpetually activated fibroblasts.
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页数:16
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