Management and prevention of stroke associated with elevated homocysteine

[31] Lindemans J(1997)Thermolabile variant of 5.10-methylenetetrahydrofolate reductase associated with low red-cell folates: implications for folate intake recommendations Lancet 349 1591-1593

[32] Coull BM(2000)Polymorphisms in the CBS gene associated with decreased risk of coronary artery disease and increased responsiveness to total homocysteine lowering by folic acid Mol Genet Metab 70 53-60

[33] Malinow MR(1999)A common mutation A1298C in human methylenetetrahydrofolate reductase gene: association with plasma total homocysteine and folate concentrations J Nutr 129 1656-1661

[34] Beamer N(1995)A quantitative assessment of plasma homocysteine as a risk factor for vascular disease. Probable benefits of increasing folic acid intake JAMA 274 1049-1071

[35] Davi G(2000)What level of plasma homocyst(e)ine should be treated? Effects of vitamin therapy on progression of carotid atherosclerosis in patients with homocyst(e)ine levels above and below 14 micromol/L Am J Hypertens 13 105-110

[36] Di Minno G(2000)Effect of homocysteine-lowering treatment with folic acid plus vitamin B6 on progression of subclinical atherosclerosis: a randomised, placebo-controlled trial Lancet 355 517-522

[37] Coppola A(1999)Endothelial dysfunction by acute hyperhomocyst(e)inemia: restoration by folic acid Clin Sci 96 235-239

[38] D’Angelo A(1999)Oral folate enhances endothelial dysfunction in hyperhomocysteinemic subjects Eur J Clin Invest 29 659-662

[39] Selhub J(2001)Decreased rate of coronary restenosis after lowering of plasma homocysteine levels N Engl J Med 345 1593-1600

[40] Cochran FB(2000)Fortification of foods with folic acid—how much is enough? N Engl J Med 342 1442-1445

[31] Lindemans J(1997)Thermolabile variant of 5.10-methylenetetrahydrofolate reductase associated with low red-cell folates: implications for folate intake recommendations Lancet 349 1591-1593

[32] Coull BM(2000)Polymorphisms in the CBS gene associated with decreased risk of coronary artery disease and increased responsiveness to total homocysteine lowering by folic acid Mol Genet Metab 70 53-60

[33] Malinow MR(1999)A common mutation A1298C in human methylenetetrahydrofolate reductase gene: association with plasma total homocysteine and folate concentrations J Nutr 129 1656-1661

[34] Beamer N(1995)A quantitative assessment of plasma homocysteine as a risk factor for vascular disease. Probable benefits of increasing folic acid intake JAMA 274 1049-1071

[35] Davi G(2000)What level of plasma homocyst(e)ine should be treated? Effects of vitamin therapy on progression of carotid atherosclerosis in patients with homocyst(e)ine levels above and below 14 micromol/L Am J Hypertens 13 105-110

[36] Di Minno G(2000)Effect of homocysteine-lowering treatment with folic acid plus vitamin B6 on progression of subclinical atherosclerosis: a randomised, placebo-controlled trial Lancet 355 517-522

[37] Coppola A(1999)Endothelial dysfunction by acute hyperhomocyst(e)inemia: restoration by folic acid Clin Sci 96 235-239

[38] D’Angelo A(1999)Oral folate enhances endothelial dysfunction in hyperhomocysteinemic subjects Eur J Clin Invest 29 659-662

[39] Selhub J(2001)Decreased rate of coronary restenosis after lowering of plasma homocysteine levels N Engl J Med 345 1593-1600

[40] Cochran FB(2000)Fortification of foods with folic acid—how much is enough? N Engl J Med 342 1442-1445