Vascular development in endometriosis

被引：167

作者：

Groothuis P.G. ^{[1
,3
]}

Nap A.W. ^{[1
]}

Winterhager E. ^{[2
]}

Grümmer R. ^{[2
]}

机构：

[1] Research Institute GROW, Department of Obstetrics and Gynaecology, University Hospital Maastricht, Maastricht

[2] Institute of Anatomy, University Hospital Duisburg-Essen, Essen

[3] Research Institute GROW, Department of Obstetrics and Gynecology, Academisch Ziekenhuis Maastricht, 6229 HX Maastricht

来源：

Angiogenesis | 2005年 / 8卷 / 2期

关键词：

Angiogenesis; Chronic inflammation; Endometriosis; Endometrium; Invasion; Migration; Peritoneum; Vasculogenesis;

D O I：

10.1007/s10456-005-9005-x

中图分类号：

学科分类号：

摘要：

Endometriosis, defined as the presence of endometrial tissue outside the uterus, is an estrogen-dependent disease which causes pelvic pain and subfertility in women of reproductive age. The condition has a dramatic impact on the professional, social and marital life of sufferers. Direct and indirect evidence suggests that angiogenesis is required for the development and persistence of endometriosis. In this review the state-of-the-art with regard to our understanding of the role of angiogenesis in the ectopic implantation and survival of menstrual endometrial tissue will be discussed. © Springer 2005.

引用

页码：147 / 156

页数：9

共 106 条

[21]

Haney A.F., Muscato J.J., Weinberg J.B., Peritoneal fluid cell populations in infertility patients, Fertil Steril, 35, 6, pp. 696-698, (1981)

[22]

Hill J.A., Faris H.M., Schiff I., Anderson D.J., Characterization of leukocyte subpopulations in the peritoneal fluid of women with endometriosis, Fertil Steril, 50, 2, pp. 216-222, (1988)

[23]

Ho H.N., Wu M.Y., Yang Y.S., Peritoneal cellular immunity and endometriosis, Am J Reprod Immunol, 38, 6, pp. 400-412, (1997)

[24]

Koninckx P.R., Is mild endometriosis a condition occurring intermittently in all women?, Hum Reprod, 9, 12, pp. 2202-2205, (1994)

[25]

Sharkey A.M., Day K., McPherson A., Et al., Vascular endothelial growth factor expression in human endometrium is regulated by hypoxia, J Clin Endocrinol Metab, 85, 1, pp. 402-409, (2000)

[26]

Harmey J.H., Dimitriadis E., Kay E., Et al., Regulation of macrophage production of vascular endothelial growth factor (VEGF) by hypoxia and transforming growth factor beta-1, Ann Surg Oncol, 5, 3, pp. 271-278, (1998)

[27]

Freeman M.R., Schneck F.X., Gagnon M.L., Et al., Peripheral blood T lymphocytes and lymphocytes infiltrating human cancers express vascular endothelial growth factor: A potential role for T cells in angiogenesis, Cancer Res, 55, 18, pp. 4140-4145, (1995)

[28]

Selgas R., Del Peso G., Bajo M.A., Et al., Spontaneous VEGF production by cultured peritoneal mesothelial cells from patients on peritoneal dialysis, Perit Dial Int, 20, 6, pp. 798-801, (2000)

[29]

Nagy J.A., Masse E.M., Herzberg K.T., Et al., Pathogenesis of ascites tumor growth: Vascular permeability factor, vascular hyperpermeability, and ascites fluid accumulation, Cancer Res, 55, 2, pp. 360-368, (1995)

[30]

Maas J.W., Calhaz-Jorge C., Ter Riet G., Dunselman G.A., Et al., Tumor necrosis factor-alpha but not interleukin-1 beta or interleukin-8 concentrations correlate with angiogenic activity of peritoneal fluid from patients with minimal to mild endometriosis, Fertil Steril, 75, 1, pp. 180-185, (2001)

← 1 2 3 4 5 6 7 8 9 10 →